5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis. - Wikidata - wikimedia - TriplyDB

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

http://www.wikidata.org/entity/Q37073396

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Serotonergic drugs and valvular heart disease Region-specific deletions of RIM1 reproduce a subset of global RIM1α(-/-) phenotypes Developmental influences on circuits programming susceptibility to obesity From neuroanatomy to behavior: central integration of peripheral signals regulating feeding behavior Brain regulation of energy balance and body weight.Central nervous control of energy and glucose balance: focus on the central melanocortin system Neural control of energy balance: translating circuits to therapies Genetics of Common Antipsychotic-Induced Adverse Effects Loss of the imprinted snoRNA mbii-52 leads to increased 5htr2c pre-RNA editing and altered 5HT2CR-mediated behaviour A serotonin and melanocortin circuit mediates D-fenfluramine anorexia c-Jun amino-terminal kinase-1 mediates glucose-responsive upregulation of the RNA editing enzyme ADAR2 in pancreatic beta-cells Serotonin 2C receptor activates a distinct population of arcuate pro-opiomelanocortin neurons via TRPC channels Ablation of Sax2 gene expression prevents diet-induced obesity Melanocortin neurons: Multiple routes to regulation of metabolism.Antiobesity pharmacotherapy: new drugs and emerging targets PI3K in the ventromedial hypothalamic nucleus mediates estrogenic actions on energy expenditure in female mice A treasure trove of hypothalamic neurocircuitries governing body weight homeostasis New central targets for the treatment of obesity.Critical determinants of hypothalamic appetitive neuropeptide development and expression: species considerations.Circuits controlling energy balance and mood: inherently intertwined or just complicated intersections?The tyrosine kinase receptor Tyro3 enhances lifespan and neuropeptide Y (Npy) neuron survival in the mouse anorexia (anx) mutation.Melanocortin-3 receptors and metabolic homeostasis.Neurobiology of inflammation-associated anorexia.Continuous 24-hour leptin, proopiomelanocortin, and amino acid measurements in human cerebrospinal fluid: correlations with plasma leptin, soluble leptin receptor, and amino acid levels Mice with altered serotonin 2C receptor RNA editing display characteristics of Prader-Willi syndrome.Effect of serotonergic anorectics on food intake and induction of Fos in brain of mice with disruption of melanocortin 3 and/or 4 receptors 5-HT obesity medication efficacy via POMC activation is maintained during aging Novel olanzapine analogues presenting a reduced H1 receptor affinity and retained 5HT2A/D2 binding affinity ratio.Dnmt3a in Sim1 neurons is necessary for normal energy homeostasis Wired on sugar: the role of the CNS in the regulation of glucose homeostasis.Serotonin receptor 2C and insulin secretion Lorcaserin and adiposopathy: 5-HT2c agonism as a treatment for 'sick fat' and metabolic disease.Fluoxetine prevents 8-OH-DPAT-induced hyperphagia in Fischer inbred rats Pathophysiology and treatment of inflammatory anorexia in chronic disease.Modulation of the central melanocortin system by leptin, insulin, and serotonin: co-ordinated actions in a dispersed neuronal network.Current pharmacotherapy for obesity: extrapolation of clinical trials data to practice.Hypothalamic inflammation: a double-edged sword to nutritional diseases.Conditional expression of Pomc in the Lepr-positive subpopulation of POMC neurons is sufficient for normal energy homeostasis and metabolism Convergence of the insulin and serotonin programs in the pancreatic β-cell Fasting activation of AgRP neurons requires NMDA receptors and involves spinogenesis and increased excitatory tone.

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5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

http://www.wikidata.org/entity/Q37073396

description

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@en

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@nl

type

label

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@en

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@nl

prefLabel

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@en

5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

@nl

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J.NEURON.2008.09.033

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5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.

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Bradford B Lowell

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Charlotte E Lee

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Daisuke Kohno

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Jason G Anderson

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Jeffrey M Zigman

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Joel K Elmquist

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Juli E Jones

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Michelle J Choi

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Yong Xu

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P2860

Mice lacking ghrelin receptors resist the development of diet-induced obesity

Eating disorder and epilepsy in mice lacking 5-HT2c serotonin receptors

Valvular heart disease associated with fenfluramine-phentermine

The snoRNA HBII-52 regulates alternative splicing of the serotonin receptor 2C

From lesions to leptin: hypothalamic control of food intake and body weight.

The role of melanocortin signalling in the control of body weight: evidence from human and murine genetic models.

Leptin differentially regulates NPY and POMC neurons projecting to the lateral hypothalamic area.

5-HT1c receptor is a prominent serotonin receptor subtype in the central nervous system

Anatomy and regulation of the central melanocortin system.

Polymorphisms of the 5-HT2C receptor and leptin genes are associated with antipsychotic drug-induced weight gain in Caucasian subjects with a first-episode psychosis.

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582-589

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4

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Kevin W. Williams

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2008-11-01T00:00:00Z

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23501998

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19038216

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2631191

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