IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
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Regulation of Neuroinflammation through Programed Death-1/Programed Death Ligand Signaling in Neurological DisordersAmyloid fibrils activate B-1a lymphocytes to ameliorate inflammatory brain diseaseFriend or foe: the dichotomous impact of T cells on neuro-de/re-generation during agingHeterogeneity of B Cell Functions in Stroke-Related Risk, Prevention, Injury, and RepairAntidepressants reduce neuroinflammatory responses and astroglial alpha-synuclein accumulation in a transgenic mouse model of multiple system atrophyRole of Interleukin-10 in Acute Brain Injuries.Mesenchymal Stem Cells Overexpressing Interleukin-10 Promote Neuroprotection in Experimental Acute Ischemic Stroke.Antigen-specific immune reactions to ischemic stroke.Brain immune cell composition and functional outcome after cerebral ischemia: comparison of two mouse strainsFunctional role of regulatory lymphocytes in stroke: facts and controversies.IL-10 producing B cells partially restore E2-mediated protection against EAE in PD-L1 deficient mice.Treatment with IL-10 producing B cells in combination with E2 ameliorates EAE severity and decreases CNS inflammation in B cell-deficient mice.PD-L1 Monoclonal Antibody Treats Ischemic Stroke by Controlling Central Nervous System InflammationRole for microglia in sex differences after ischemic stroke: importance of M2Estrogen induces multiple regulatory B cell subtypes and promotes M2 microglia and neuroprotection during experimental autoimmune encephalomyelitisPartial MHC Constructs Treat Thromboembolic Ischemic Stroke Characterized by Early Immune ExpansionProtective effect of Xuebijing injection against acute lung injury induced by left ventricular ischemia/reperfusion in rabbits.Splenic responses in ischemic stroke: new insights into stroke pathology.Effects of Stroke on the Autonomic Nervous System.Role of Circulating Immune Cells in Stroke and Preconditioning-Induced Protection.Amelioration of Ischemic Brain Injury in Rats With Human Umbilical Cord Blood Stem Cells: Mechanisms of Action.Regulatory T Cells in Post-stroke Immune Homeostasis.Critical Role of the Sphingolipid Pathway in Stroke: a Review of Current Utility and Potential Therapeutic Targets.Repetitive hypoxic preconditioning induces an immunosuppressed B cell phenotype during endogenous protection from stroke.Preconditioning-induced CXCL12 upregulation minimizes leukocyte infiltration after stroke in ischemia-tolerant mice.Stroke induces a rapid adaptive autoimmune response to novel neuronal antigens.Pre-existing interleukin 10 in cerebral arteries attenuates subsequent brain injury caused by ischemia/reperfusion.Treatment of experimental stroke with IL-10-producing B-cells reduces infarct size and peripheral and CNS inflammation in wild-type B-cell-sufficient mice.Regulatory CD8(+)CD122 (+) T-cells predominate in CNS after treatment of experimental stroke in male mice with IL-10-secreting B-cells.Pivotal neuroinflammatory and therapeutic role of high mobility group box 1 in ischemic stroke.Brain-immune interactions in perinatal hypoxic-ischemic brain injury.Sex differences in regulatory cells in experimental stroke.Regulatory B cells in Experimental Stroke.Role of interleukin-10 in the neuroprotective effect of the Angiotensin Type 2 Receptor agonist, compound 21, after ischemia/reperfusion injury.The role of dopaminergic immune cell signalling in poststroke inflammation.Splenic responses play an important role in remote ischemic preconditioning-mediated neuroprotection against stroke.
P2860
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P2860
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@en
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@nl
type
label
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@en
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@nl
prefLabel
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@en
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@nl
P2093
P2860
P1476
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
@en
P2093
Arthur A Vandenbark
Halina Offner
Sheetal Bodhankar
Stephanie J Murphy
Yingxin Chen
P2860
P2888
P304
P356
10.1007/S11011-013-9413-3
P577
2013-05-03T00:00:00Z