Design of mutant beta2 subunits as decoy molecules to reduce the expression of functional Ca2+ channels in cardiac cells.
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Ion channel remodeling in vascular smooth muscle during hypertension: Implications for novel therapeutic approachesThe ß subunit of voltage-gated Ca2+ channelsOverexpression of the Large-Conductance, Ca2+-Activated K+ (BK) Channel Shortens Action Potential Duration in HL-1 CardiomyocytesThe β3 subunit contributes to vascular calcium channel upregulation and hypertension in angiotensin II-infused C57BL/6 mice.Modification of cardiovascular ion channels by gene therapy.Cav1.2, cell proliferation, and new target in atherosclerosis.Delivery of ion channel genes to treat cardiovascular diseases.Use of escin as a perforating agent on the IonWorks quattro automated electrophysiology platform.Prenatal Hypoxia Induced Dysfunction in Cerebral Arteries of Offspring Rats.
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Design of mutant beta2 subunits as decoy molecules to reduce the expression of functional Ca2+ channels in cardiac cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 January 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@en
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
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type
label
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@en
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@nl
prefLabel
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@en
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@nl
P2093
P2860
P356
P1476
Design of mutant beta2 subunit ...... a2+ channels in cardiac cells.
@en
P2093
James D Marsh
Joseph R Stimers
Nancy J Rusch
Sabine Télémaque
Sung W Rhee
Swapnil Sonkusare
Terrie Grain
P2860
P356
10.1124/JPET.107.128215
P407
P577
2008-01-09T00:00:00Z