Prolonged treatment of porcine pulmonary artery with nitric oxide decreases cGMP sensitivity and cGMP-dependent protein kinase specific activity
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Increased degradation of MYPT1 contributes to the development of tolerance to nitric oxide in porcine pulmonary artery.Transcriptional and post-transcriptional regulation of cGMP-dependent protein kinase (PKG-I): pathophysiological significanceDifferential roles of cAMP and cGMP in megakaryocyte maturation and platelet biogenesis.Newborn rat response to single vs. combined cGMP-dependent pulmonary vasodilators.
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Prolonged treatment of porcine pulmonary artery with nitric oxide decreases cGMP sensitivity and cGMP-dependent protein kinase specific activity
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 October 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Prolonged treatment of porcine ...... otein kinase specific activity
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Prolonged treatment of porcine ...... tein kinase specific activity.
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Prolonged treatment of porcine ...... otein kinase specific activity
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Prolonged treatment of porcine ...... tein kinase specific activity.
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Prolonged treatment of porcine ...... otein kinase specific activity
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Prolonged treatment of porcine ...... tein kinase specific activity.
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Prolonged treatment of porcine ...... otein kinase specific activity
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David O Warner
Keith A Jones
William J Perkins
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10.1152/AJPLUNG.90318.2008
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2008-10-24T00:00:00Z