Motoneuron BDNF/TrkB signaling enhances functional recovery after cervical spinal cord injury.
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Neuroprotective and Neurorestorative Processes after Spinal Cord Injury: The Case of the Bulbospinal Respiratory NeuronsGene delivery strategies to promote spinal cord repairPAR2-mediated upregulation of BDNF contributes to central sensitization in bone cancer pain.TrkB kinase activity is critical for recovery of respiratory function after cervical spinal cord hemisection.Biocompatibility of a coacervate-based controlled release system for protein delivery to the injured spinal cordLocalized delivery of brain-derived neurotrophic factor-expressing mesenchymal stem cells enhances functional recovery following cervical spinal cord injury.Phrenic long-term facilitation requires PKCθ activity within phrenic motor neurons.Impact of unilateral denervation on transdiaphragmatic pressure.TrkB gene therapy by adeno-associated virus enhances recovery after cervical spinal cord injury.Transporter Protein-Coupled DPCPX Nanoconjugates Induce Diaphragmatic Recovery after SCI by Blocking Adenosine A1 Receptors.Functional impact of sarcopenia in respiratory muscles.Intravenous administration of adipose tissue-derived stem cells enhances nerve healing and promotes BDNF expression via the TrkB signaling in a rat stroke modelFailed reinnervation in aging skeletal muscle.Long-term viral brain-derived neurotrophic factor delivery promotes spasticity in rats with a cervical spinal cord hemisection.Tissue engineering is a promising method for the repair of spinal cord injuries (Review)Neurotrophins and spinal circuit function.Cellular transplantation-based evolving treatment options in spinal cord injury.Functional recovery after cervical spinal cord injury: Role of neurotrophin and glutamatergic signaling in phrenic motoneurons.The Impact of Midcervical Contusion Injury on Diaphragm Muscle Function.Plasticity in respiratory motor neurons in response to reduced synaptic inputs: A form of homeostatic plasticity in respiratory control?Motoneuron glutamatergic receptor expression following recovery from cervical spinal hemisection.The crossed phrenic phenomenon.Local Injection of Lenti-BDNF at the Lesion Site Promotes M2 Macrophage Polarization and Inhibits Inflammatory Response After Spinal Cord Injury in Mice.Functional Measurement of Respiratory Muscle Motor Behaviors Using Transdiaphragmatic Pressure.A murine model of cervical spinal cord injury to study post-lesional respiratory neuroplasticity.Phrenic motor neuron adenosine 2A receptors elicit phrenic motor facilitation.Diaphragm muscle activity across respiratory motor behaviors in awake and lightly anesthetized rats.Intermittent hypoxia promotes recovery of respiratory motor function in spinal cord-injured mice depleted of serotonin in the central nervous system.Impact of glutamatergic and serotonergic neurotransmission on diaphragm muscle activity after cervical spinal hemisection.High-frequency epidural stimulation across the respiratory cycle evokes phrenic short-term potentiation after incomplete cervical spinal cord injury.Electro-acupuncture at Governor Vessel improves neurological function in rats with spinal cord injury.Whole body vibration (WBV) following spinal cord injury (SCI) in rats: Timing of intervention.Diaphragm electromyographic activity following unilateral midcervical contusion injury in rats.BDNF effects on functional recovery across motor behaviors after cervical spinal cord injury.Intraspinal transplantation of subventricular zone-derived neural progenitor cells improves phrenic motor output after high cervical spinal cord injury.Phrenic motor neuron TrkB expression is necessary for acute intermittent hypoxia-induced phrenic long-term facilitation.Sustained-release of FGF-2 from a hybrid hydrogel of heparin-poloxamer and decellular matrix promotes the neuroprotective effects of proteins after spinal injury.Gelatin Nanostructured Lipid Carriers Incorporating Nerve Growth Factor Inhibit Endoplasmic Reticulum Stress-Induced Apoptosis and Improve Recovery in Spinal Cord Injury.Acute intermittent hypoxia and rehabilitative training following cervical spinal injury alters neuronal hypoxia- and plasticity-associated protein expression.
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Motoneuron BDNF/TrkB signaling enhances functional recovery after cervical spinal cord injury.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 10 April 2013
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@en
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@nl
type
label
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@en
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@nl
prefLabel
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@en
Motoneuron BDNF/TrkB signaling ...... r cervical spinal cord injury.
@nl
P2860
P50
P1476
Motoneuron BDNF/TrkB signaling ...... er cervical spinal cord injury
@en
P2093
Wen-Zhi Zhan
P2860
P304
P356
10.1016/J.EXPNEUROL.2013.04.002
P407
P577
2013-04-10T00:00:00Z