Reexpression of pyruvate kinase M2 in type 1 myofibers correlates with altered glucose metabolism in myotonic dystrophy.
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Abnormal splicing switch of DMD's penultimate exon compromises muscle fibre maintenance in myotonic dystrophy.Expanded CCUG repeat RNA expression in Drosophila heart and muscle trigger Myotonic Dystrophy type 1-like phenotypes and activate autophagocytosis genes.Autoregulation of MBNL1 function by exon 1 exclusion from MBNL1 transcript.Mitochondrial integrity in a neonatal bovine model of right ventricular dysfunctionA PKM2 signature in the failing heart.c-Myc Alters Substrate Utilization and O-GlcNAc Protein Posttranslational Modifications without Altering Cardiac Function during Early Aortic ConstrictionCUG-BP1 regulates RyR1 ASI alternative splicing in skeletal muscle atrophy.The impact of RNA binding motif protein 4-regulated splicing cascade on the progression and metabolism of colorectal cancer cells.RBM4a-regulated splicing cascade modulates the differentiation and metabolic activities of brown adipocytes.RNA-binding protein CELF1 promotes tumor growth and alters gene expression in oral squamous cell carcinoma.Identification of CELF1 RNA targets by CLIP-seq in human HeLa cellsAgeing has no effect on the regulation of the ubiquitin proteasome-related genes and proteins following resistance exercise.The genetic basis for individual differences in mRNA splicing and APOBEC1 editing activity in murine macrophages.Developmental insights into the pathology of and therapeutic strategies for DM1: Back to the basics.Can Human Pluripotent Stem Cell-Derived Cardiomyocytes Advance Understanding of Muscular Dystrophies?Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylationReceptor and post-receptor abnormalities contribute to insulin resistance in myotonic dystrophy type 1 and type 2 skeletal muscle.Emerging roles of RNA-binding proteins in diabetes and their therapeutic potential in diabetic complications.AMPKα1-LDH pathway regulates muscle stem cell self-renewal by controlling metabolic homeostasis.Altered levels of the splicing factor muscleblind modifies cerebral cortical function in mouse models of myotonic dystrophy.Protein kinase C theta (PKCθ) modulates the ClC-1 chloride channel activity and skeletal muscle phenotype: a biophysical and gene expression study in mouse models lacking the PKCθ.Transcriptional profiling reveals extraordinary diversity among skeletal muscle tissues.miR-23b and miR-218 silencing increase Muscleblind-like expression and alleviate myotonic dystrophy phenotypes in mammalian models.
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P2860
Reexpression of pyruvate kinase M2 in type 1 myofibers correlates with altered glucose metabolism in myotonic dystrophy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 30 July 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@en
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@nl
type
label
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@en
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@nl
prefLabel
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@en
Reexpression of pyruvate kinas ...... abolism in myotonic dystrophy.
@nl
P2860
P356
P1476
Reexpression of pyruvate kinas ...... tabolism in myotonic dystrophy
@en
P2093
Thomas A Cooper
P2860
P304
13570-13575
P356
10.1073/PNAS.1308806110
P407
P50
P577
2013-07-30T00:00:00Z