Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein. - Wikidata - wikimedia - TriplyDB

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

http://www.wikidata.org/entity/Q37104090

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HIV-1 Tat exacerbates lipopolysaccharide-induced cytokine release via TLR4 signaling in the enteric nervous system.Neurological immune reconstitution inflammatory response: riding the tide of immune recovery Morphine enhances HIV-1SF162-mediated neuron death and delays recovery of injured neurites Platelet Activation in Human Immunodeficiency Virus Type-1 Patients Is Not Altered with Cocaine Abuse HIV-1 transgenic female rat: synaptodendritic alterations of medium spiny neurons in the nucleus accumbens.The use of Nanotrap particles technology in capturing HIV-1 virions and viral proteins from infected cells.Three-dimensional imaging of HIV-1 virological synapses reveals membrane architectures involved in virus transmission.HIV-1 Tat-induced changes in synaptically-driven network activity adapt during prolonged exposure.STAT3 and its phosphorylation are involved in HIV-1 Tat-induced transactivation of glial fibrillary acidic protein HIV-1-Tat excites cardiac parasympathetic neurons of nucleus ambiguus and triggers prolonged bradycardia in conscious rats.Astrocyte elevated gene-1 is a novel modulator of HIV-1-associated neuroinflammation via regulation of nuclear factor-κB signaling and excitatory amino acid transporter-2 repression.HIV-1 Tat inhibits EAAT-2 through AEG-1 upregulation in models of HIV-associated neurocognitive disorder.RNA-directed gene editing specifically eradicates latent and prevents new HIV-1 infection.Challenges in HIV Vaccine Research for Treatment and Prevention.Interaction between HIV-1 Tat and DNA-PKcs modulates HIV transcription and class switch recombination.Effects of HIV-1 Tat on enteric neuropathogenesis.New insights into immune reconstitution inflammatory syndrome of the central nervous system.Caffeine Blocks HIV-1 Tat-Induced Amyloid Beta Production and Tau Phosphorylation.Ibudilast (AV411), and its AV1013 analog, reduce HIV-1 replication and neuronal death induced by HIV-1 and morphine Effects of chronic HIV-1 Tat exposure in the CNS: heightened vulnerability of males versus females to changes in cell numbers, synaptic integrity, and behavior HIV-1 Tat C phosphorylates VE-cadherin complex and increases human brain microvascular endothelial cell permeability.Creatine protects against mitochondrial dysfunction associated with HIV-1 Tat-induced neuronal injury.Sensitization of enteric neurons to morphine by HIV-1 Tat protein.HIV-1 Tat immunization restores immune homeostasis and attacks the HAART-resistant blood HIV DNA: results of a randomized phase II exploratory clinical trial Eradication of human immunodeficiency virus from brain reservoirs.HIV eradication symposium: will the brain be left behind?Oligodendrocytes Are Targets of HIV-1 Tat: NMDA and AMPA Receptor-Mediated Effects on Survival and Development.HIV-1 Tat protein vaccination in mice infected with Mycobacterium tuberculosis is safe, immunogenic and reduces bacterial lung pathology.Chronic HIV-1 Tat and HIV reduce Rbfox3/NeuN: evidence for sex-related effects HIV-1-Tat Protein Inhibits SC35-mediated Tau Exon 10 Inclusion through Up-regulation of DYRK1A Kinase.HIV-Tat immunization induces cross-clade neutralizing antibodies and CD4(+) T cell increases in antiretroviral-treated South African volunteers: a randomized phase II clinical trial HIV-1 Tat Regulates Occludin and Aβ Transfer Receptor Expression in Brain Endothelial Cells via Rho/ROCK Signaling Pathway Update on HIV-associated neurocognitive disorders.HIV-1 Tat-shortened neurite outgrowth through regulation of microRNA-132 and its target gene expression.HIV-1 Tat Inhibits Autotaxin Lysophospholipase D Activity and Modulates Oligodendrocyte Differentiation Central nervous system immune reconstitution inflammatory syndrome.HIV Tat Impairs Neurogenesis through Functioning As a Notch Ligand and Activation of Notch Signaling Pathway.NMDARs Adapt to Neurotoxic HIV Protein Tat Downstream of a GluN2A-Ubiquitin Ligase Signaling Pathway.Novel neuroprotective GSK-3β inhibitor restricts Tat-mediated HIV-1 replication.Synaptodendritic injury with HIV-Tat protein: What is the therapeutic target?

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Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

http://www.wikidata.org/entity/Q37104090

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 July 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@en

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@nl

type

label

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@en

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@nl

prefLabel

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@en

Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

@nl

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PNAS.1308673110

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Proceedings of the National Academy of Sciences of the United States of America

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Induction of IL-17 and nonclassical T-cell activation by HIV-Tat protein.

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Avindra Nath

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Dragan Maric

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Karan Patel

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Kory R Johnson

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Rodrigo Hasbun

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Tory P Johnson

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P2860

Clinical features and serum biomarkers in HIV immune reconstitution inflammatory syndrome after cryptococcal meningitis: a prospective cohort study

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

HIV-1 Tat enters T cells using coated pits before translocating from acidified endosomes and eliciting biological responses

Diversity of IL-17-producing T lymphocytes

Therapeutic immunization with HIV-1 Tat reduces immune activation and loss of regulatory T-cells and improves immune function in subjects on HAART

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Proinflammatory cytokines regulate antigen-independent T-cell activation by two separate calcium-signaling pathways in multiple sclerosis patients.

Progression to the G1b phase of the cell cycle is required for completion of human immunodeficiency virus type 1 reverse transcription in T cells.

Active chromatin domains are defined by acetylation islands revealed by genome-wide mapping

Relapsing and remitting human immunodeficiency virus-associated leukoencephalomyelopathy.

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10.1073/PNAS.1308673110

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33

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Peter A Calabresi

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2013-07-29T00:00:00Z

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