Calcium-activated potassium channels contribute to human skeletal muscle microvascular endothelial dysfunction related to cardiopulmonary bypass.
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Naphtho[1,2-d]thiazol-2-ylamine (SKA-31), a new activator of KCa2 and KCa3.1 potassium channels, potentiates the endothelium-derived hyperpolarizing factor response and lowers blood pressureRegulation of Microvascular Function by Adipose Tissue in Obesity and Type 2 Diabetes: Evidence of an Adipose-Vascular Loop.Endothelin-1-induced contractile responses of human coronary arterioles via endothelin-A receptors and PKC-alpha signaling pathways.Protection of coronary endothelial function during cardiac surgery: potential of targeting endothelial ion channels in cardioprotectionDecreased contractile response to endothelin-1 of peripheral microvasculature from diabetic patients.Improvement of endothelium-dependent vasodilations by SKA-31 and SKA-20, activators of small- and intermediate-conductance Ca2+ -activated K+ -channels.Hyperhomocysteinemia impairs endothelium-derived hyperpolarizing factor-mediated vasorelaxation in transgenic cystathionine beta synthase-deficient micePharmacological activation of KCa3.1/KCa2.3 channels produces endothelial hyperpolarization and lowers blood pressure in conscious dogsInactivation of Endothelial Small/Intermediate Conductance of Calcium-Activated Potassium Channels Contributes to Coronary Arteriolar Dysfunction in Diabetic Patients.Changes in microvascular reactivity after cardiopulmonary bypass in patients with poorly controlled versus controlled diabetes.Large conductance calcium-activated potassium channels contribute to the reduced myogenic tone of peripheral microvasculature after cardiopulmonary bypass.Vascular KCa-channels as therapeutic targets in hypertension and restenosis disease.Endothelial Ca+-activated K+ channels in normal and impaired EDHF-dilator responses--relevance to cardiovascular pathologies and drug discoveryCalcium-activated potassium channels - a therapeutic target for modulating nitric oxide in cardiovascular disease?Nitric oxide: orchestrator of endothelium-dependent responses.Microvascular dysfunction in patients with diabetes after cardioplegic arrest and cardiopulmonary bypass.Diabetes and Cardioplegia.Role of Nrf2 Signaling in the Regulation of Vascular BK Channel β1 Subunit Expression and BK Channel Function in High-Fat Diet-Induced Diabetic Mice.Decreased coronary arteriolar response to KCa channel opener after cardioplegic arrest in diabetic patients.
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P2860
Calcium-activated potassium channels contribute to human skeletal muscle microvascular endothelial dysfunction related to cardiopulmonary bypass.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on August 2008
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@en
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
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type
label
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@en
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@nl
prefLabel
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@en
Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@nl
P2093
P2860
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Calcium-activated potassium ch ...... ted to cardiopulmonary bypass.
@en
P2093
Eric W Sellke
Frank W Sellke
Kamal R Khabbaz
Neel R Sodha
Richard T Clements
Seth L Alper
Venkatachalam Senthilnathan
Yuhong Liu
P2860
P304
P356
10.1016/J.SURG.2008.03.032
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P577
2008-08-01T00:00:00Z