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Hsp72 overexpression accelerates the recovery from caerulein-induced pancreatitisHeat shock protein 27 as a prognostic and predictive biomarker in pancreatic ductal adenocarcinoma.Separation and identification of HSP-associated protein complexes from pancreatic cancer cell lines using 2D CN/SDS-PAGE coupled with mass spectrometry.Serum HSP70: a novel biomarker for early detection of pancreatic cancer.MUC1c regulates cell survival in pancreatic cancer by preventing lysosomal permeabilization.Second-line rescue therapy of helicobacter pylori infection.Down-regulation of HSP60 expression by RNAi increases lipopolysaccharide- and cerulein-induced damages on isolated rat pancreatic tissues.Primary and liver metastasis-derived cell lines from KrasG12D; Trp53R172H; Pdx-1 Cre animals undergo apoptosis in response to triptolide.Heat shock proteins and autophagy in rats with cerulein-induced acute pancreatitisComparison of sequential therapy and amoxicillin/tetracycline containing bismuth quadruple therapy for the first-line eradication of Helicobacter pylori: a prospective, multi-center, randomized clinical trial.Triptolide inhibits MDM2 and induces apoptosis in acute lymphoblastic leukemia cells through a p53-independent pathway.Protease activation during in vivo pancreatitis is dependent on calcineurin activation.Effect of Previous High Glutamine Infusion on Inflammatory Mediators and Mortality in an Acute Pancreatitis Model.Cancer biomarkers for hepatocellular carcinomas: from traditional markers to recent topics.Acute pancreatitis: the stress factor.Chaperoning proteins for destruction: diverse roles of Hsp70 chaperones and their co-chaperones in targeting misfolded proteins to the proteasomeTherapeutic potential of laminin-biodritin microcapsules for type 1 diabetes mellitus.Prosurvival role of heat shock factor 1 in the pathogenesis of pancreatobiliary tumors.Heat shock protein 72 protects kidney proximal tubule cells from injury induced by triptolide by means of activation of the MEK/ERK pathway.ACG Clinical Guideline: Treatment of Helicobacter pylori Infection.Rescue Therapy for Helicobacter pylori Eradication: A Randomized Non-Inferiority Trial of Amoxicillin or Tetracycline in Bismuth Quadruple Therapy.Protective effects of heme oxygenase-1 against severe acute pancreatitis via inhibition of tumor necrosis factor-α and augmentation of interleukin-10.Copper or/and arsenic induce oxidative stress-cascaded, nuclear factor kappa B-dependent inflammation and immune imbalance, trigging heat shock response in the kidney of chicken.Ibuprofen and diclofenac treatments reduce proliferation of pancreatic acinar cells upon inflammatory injury and mitogenic stimulation.Third-line rescue therapy with bismuth-containing quadruple regimen after failure of two treatments (with clarithromycin and levofloxacin) for H. pylori infection.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Heat shock proteins in pancreatic diseases.
@en
Heat shock proteins in pancreatic diseases.
@nl
type
label
Heat shock proteins in pancreatic diseases.
@en
Heat shock proteins in pancreatic diseases.
@nl
prefLabel
Heat shock proteins in pancreatic diseases.
@en
Heat shock proteins in pancreatic diseases.
@nl
P2860
P1476
Heat shock proteins in pancreatic diseases.
@en
P2093
Ashok Saluja
Vikas Dudeja
P2860
P356
10.1111/J.1440-1746.2007.05272.X
P478
23 Suppl 1
P577
2008-03-01T00:00:00Z