Upregulated WDR26 serves as a scaffold to coordinate PI3K/ AKT pathway-driven breast cancer cell growth, migration, and invasion.
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Cell signalling pathway regulation by RanBPM: molecular insights and disease implications.A statistical method for detecting differentially expressed SNVs based on next-generation RNA-seq data.Characterization of Novel Molecular Mechanisms Favoring Rac1 Membrane Translocation.Combretastatin A4 Regulates Proliferation, Migration, Invasion, and Apoptosis of Thyroid Cancer Cells via PI3K/Akt Signaling Pathway.Inhibiting G protein βγ signaling blocks prostate cancer progression and enhances the efficacy of paclitaxel.Angiotensin-(1-7) counteracts the transforming effects triggered by angiotensin II in breast cancer cells.WDR26 Haploinsufficiency Causes a Recognizable Syndrome of Intellectual Disability, Seizures, Abnormal Gait, and Distinctive Facial Features.MicroRNA‑21 increases cell viability and suppresses cellular apoptosis in non‑small cell lung cancer by regulating the PI3K/Akt signaling pathway.Triangle of AKT2, miRNA, and Tumorigenesis in Different Cancers.MiR-29b-1-5p is altered in BRCA1 mutant tumours and is a biomarker in basal-like breast cancer
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Upregulated WDR26 serves as a scaffold to coordinate PI3K/ AKT pathway-driven breast cancer cell growth, migration, and invasion.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 17 February 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Upregulated WDR26 serves as a ...... owth, migration, and invasion.
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Upregulated WDR26 serves as a ...... owth, migration, and invasion.
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type
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Upregulated WDR26 serves as a ...... owth, migration, and invasion.
@en
Upregulated WDR26 serves as a ...... owth, migration, and invasion.
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prefLabel
Upregulated WDR26 serves as a ...... owth, migration, and invasion.
@en
Upregulated WDR26 serves as a ...... owth, migration, and invasion.
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P2860
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P1433
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Upregulated WDR26 serves as a ...... owth, migration, and invasion.
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Xiaoyun Tang
Yuanchao Ye
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P304
17854-17869
P356
10.18632/ONCOTARGET.7439
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P577
2016-02-17T00:00:00Z