Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy - Wikidata - wikimedia - TriplyDB

Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy

Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy

http://www.wikidata.org/entity/Q37110591

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MicroRNAs in Diabetic Kidney Disease Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases MicroRNA-23b Targets Ras GTPase-Activating Protein SH3 Domain-Binding Protein 2 to Alleviate Fibrosis and Albuminuria in Diabetic Nephropathy Systems biology combining human- and animal-data miRNA and mRNA data identifies new targets in ureteropelvic junction obstruction The role of microRNAs in diabetic nephropathy.Transforming growth factor β1 (TGF-β1) enhances expression of profibrotic genes through a novel signaling cascade and microRNAs in renal mesangial cells MicroRNAs and their applications in kidney diseases Hyperglycemia, p53, and mitochondrial pathway of apoptosis are involved in the susceptibility of diabetic models to ischemic acute kidney injury.Role of microRNA 1207-5P and its host gene, the long non-coding RNA Pvt1, as mediators of extracellular matrix accumulation in the kidney: implications for diabetic nephropathy MicroRNAs in renal fibrosis.Combination of active components of Xiexin decoction ameliorates renal fibrosis through the inhibition of NF-κB and TGF-β1/Smad pathways in db/db diabetic mice Integrated genomic analysis identifies subclasses and prognosis signatures of kidney cancer.Epigenetic mechanisms of endothelial dysfunction in type 2 diabetes.Sirtuin 1: A Target for Kidney Diseases.Circulating TGF-β1-Regulated miRNAs and the Risk of Rapid Progression to ESRD in Type 1 Diabetes.Comparison of different normalization strategies for the analysis of glomerular microRNAs in IgA nephropathy.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.Cordyceps militaris Treatment Preserves Renal Function in Type 2 Diabetic Nephropathy Mice.Diabetic nephropathy: What does the future hold?The Expression of miR-192 and Its Significance in Diabetic Nephropathy Patients with Different Urine Albumin Creatinine Ratio.Prediction of the molecular mechanisms and potential therapeutic targets for diabetic nephropathy by bioinformatics methods.Corosolic acid inhibits the proliferation of glomerular mesangial cells and protects against diabetic renal damage p66Shc: A novel biomarker of tubular oxidative injury in patients with diabetic nephropathy.MicroRNAs: potential mediators and biomarkers of diabetic complications.MicroRNA and nephropathy: emerging concepts.Epigenetic Regulations in Diabetic Nephropathy.New insights into p53 functions through its target microRNAs.Role of microRNA machinery in kidney fibrosis.Research progress in signalling pathway in diabetic nephropathy.Diabetic nephropathy--emerging epigenetic mechanisms.RNA expression signatures and posttranscriptional regulation in diabetic nephropathy.microRNAs in Diabetic Kidney Disease.MicroRNAs as Master Regulators of Glomerular Function in Health and Disease.Epigenetics of kidney disease.Role of biomarkers in predicting diabetes complications with special reference to diabetic foot ulcers.An endoplasmic reticulum stress-regulated lncRNA hosting a microRNA megacluster induces early features of diabetic nephropathy.Curcumin protects rat liver from streptozotocin-induced diabetic pathophysiology by counteracting reactive oxygen species and inhibiting the activation of p53 and MAPKs mediated stress response pathways.Anti-Inflammatory Role of MicroRNA-146a in the Pathogenesis of Diabetic Nephropathy.Wild-type and mutant p53 differentially modulate miR-124/iASPP feedback following pohotodynamic therapy in human colon cancer cell line.MicroRNA-192 regulates hypertrophic scar fibrosis by targeting SIP1.

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Transforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathy

http://www.wikidata.org/entity/Q37110591

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 May 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Transforming growth factor-β-i ...... enesis of diabetic nephropathy

@en

Transforming growth factor-β-i ...... nesis of diabetic nephropathy.

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type

label

Transforming growth factor-β-i ...... enesis of diabetic nephropathy

@en

Transforming growth factor-β-i ...... nesis of diabetic nephropathy.

@nl

prefLabel

Transforming growth factor-β-i ...... enesis of diabetic nephropathy

@en

Transforming growth factor-β-i ...... nesis of diabetic nephropathy.

@nl

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Transforming growth factor-β-i ...... enesis of diabetic nephropathy

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Jennifer Y Lai

http://www.w3.org/2001/XMLSchema#string

Linda L Lanting

http://www.w3.org/2001/XMLSchema#string

Markus Bitzer

http://www.w3.org/2001/XMLSchema#string

Mei Wang

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Mitsuo Kato

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Rama Natarajan

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Robert G Nelson

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Sumanth Putta

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Supriya D Deshpande

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P2860

MicroRNAs: target recognition and regulatory functions

Downregulation of p53-inducible microRNAs 192, 194, and 215 impairs the p53/MDM2 autoregulatory loop in multiple myeloma development

MicroRNAs in stress signaling and human disease

Improved elucidation of biological processes linked to diabetic nephropathy by single probe-based microarray data analysis

Activation of p53 promotes renal injury in acute aristolochic acid nephropathy.

Systematic analysis of a novel human renal glomerulus-enriched gene expression dataset.

Loss of MicroRNA-192 promotes fibrogenesis in diabetic nephropathy

E-cadherin expression is regulated by miR-192/215 by a mechanism that is independent of the profibrotic effects of transforming growth factor-beta

MicroRNA-34a is induced via p53 during cisplatin nephrotoxicity and contributes to cell survival.

miR-192 mediates TGF-beta/Smad3-driven renal fibrosis.

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3151-3162

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scholarly article

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10.2337/DB13-0305

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9

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62

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2013-05-06T00:00:00Z

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236643242

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23649518

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3749352

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