Control of the innate immune response by the mevalonate pathway. - Wikidata - wikimedia - TriplyDB

Control of the innate immune response by the mevalonate pathway.

Control of the innate immune response by the mevalonate pathway.

http://www.wikidata.org/entity/Q37114315

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Rice Bran and Probiotics Alter the Porcine Large Intestine and Serum Metabolomes for Protection against Human Rotavirus Diarrhea The Yersinia pestis Effector YopM Inhibits Pyrin Inflammasome Activation.Familial Mediterranean fever mutations lift the obligatory requirement for microtubules in Pyrin inflammasome activation.Genetic and Epigenetic Determinants in Autoinflammatory Diseases.The Intracellular Cholesterol Landscape: Dynamic Integrator of the Immune Response.Mechanisms governing inflammasome activation, assembly and pyroptosis induction.Posttranslational Modification as a Critical Determinant of Cytoplasmic Innate Immune Recognition.The monogenic autoinflammatory diseases define new pathways in human innate immunity and inflammation.Open-Label, Phase II Study to Assess the Efficacy and Safety of Canakinumab Treatment in Active Hyperimmunoglobulinemia D With Periodic Fever Syndrome.Metabolism in Immune Cell Differentiation and Function.IL-1β and Caspase-1 Drive Autoinflammatory Disease Independently of IL-1α or Caspase-8 in a Mouse Model of Familial Mediterranean Fever.Inflammasome assembly in the chorioamniotic membranes during spontaneous labor at term.The Yersinia Virulence Factor YopM Hijacks Host Kinases to Inhibit Type III Effector-Triggered Activation of the Pyrin Inflammasome.KRASG12D expression in lung-resident myeloid cells promotes pulmonary LCH-like neoplasm sensitive to statin treatment.A dRAStic RHOAdblock of Pyrin inflammasome activation.Mevalonate metabolism governs cancer immune surveillance.Cell size control - a mechanism for maintaining fitness and function.Inflammasome activation and assembly at a glance.Recent advances in inflammasome biology.Familial Mediterranean fever mutations are hypermorphic mutations that specifically decrease the activation threshold of the Pyrin inflammasome.COPs and POPs Patrol Inflammasome Activation.Function and mechanism of the pyrin inflammasome.Mitochondrial Acetyl-CoA Synthetase 3 is Biosignature of Gastric Cancer Progression.Perturbation of Intracellular Cholesterol and Fatty Acid Homeostasis During Flavivirus Infections.

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Control of the innate immune response by the mevalonate pathway.

http://www.wikidata.org/entity/Q37114315

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 06 June 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Control of the innate immune response by the mevalonate pathway.

@en

Control of the innate immune response by the mevalonate pathway.

@nl

type

label

Control of the innate immune response by the mevalonate pathway.

@en

Control of the innate immune response by the mevalonate pathway.

@nl

prefLabel

Control of the innate immune response by the mevalonate pathway.

@en

Control of the innate immune response by the mevalonate pathway.

@nl

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Nature Immunology

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Control of the innate immune response by the mevalonate pathway

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Annie S Li

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Celia E Foster

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Charles V Rosadini

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Daniel L Kastner

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David Miao

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Donghai Wang

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Douglas T Golenbock

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Ellen M Gravallese

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Jae Jin Chae

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Jonathan C Kagan

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P2860

Binding of ras to phosphoinositide 3-kinase p110alpha is required for ras-driven tumorigenesis in mice

Glycogen synthase kinase 3: a point of convergence for the host inflammatory response

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

The kinase Akt1 controls macrophage response to lipopolysaccharide by regulating microRNAs

Pattern recognition receptors and inflammation

Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

Signaling by the phosphoinositide 3-kinase family in immune cells

B-cell adaptor for PI3K (BCAP) negatively regulates Toll-like receptor signaling through activation of PI3K

A promiscuous lipid-binding protein diversifies the subcellular sites of toll-like receptor signal transduction.

The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism

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922-929

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scholarly article

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10.1038/NI.3487

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8

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17

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2016-06-06T00:00:00Z

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27270400

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4955724

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