Impaired apoptotic cell clearance in CGD due to altered macrophage programming is reversed by phosphatidylserine-dependent production of IL-4
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Identification of a novel macrophage phenotype that develops in response to atherogenic phospholipids via Nrf2.Apoptotic cell clearance: basic biology and therapeutic potential.Resolution of inflammation: an integrated viewEmerging roles for lysophosphatidylserine in resolution of inflammationMechanism-Based Strategies for the Management of Autoimmunity and Immune Dysregulation in Primary ImmunodeficienciesNeutrophils regulate tissue Neutrophilia in inflammation via the oxidant-modified lipid lysophosphatidylserineSignaling via macrophage G2A enhances efferocytosis of dying neutrophils by augmentation of Rac activity.Monocyte- and macrophage-targeted NADPH oxidase mediates antifungal host defense and regulation of acute inflammation in mice.Alternative activation of macrophages by IL-4 impairs phagocytosis of pathogens but potentiates microbial-induced signalling and cytokine secretionNeuroendocrine signaling via the serotonin transporter regulates clearance of apoptotic cells.Programmed cell clearance: molecular regulation of the elimination of apoptotic cell corpses and its role in the resolution of inflammation.NOX2-dependent regulation of inflammationT cell-macrophage interactions and granuloma formation in vasculitis.Obesity impairs apoptotic cell clearance in asthma.PPARγ activation normalizes resolution of acute sterile inflammation in murine chronic granulomatous disease.γ-Interferon-inducible lysosomal thiol reductase (GILT) maintains phagosomal proteolysis in alternatively activated macrophagesMacrophage-specific NOX2 contributes to the development of lung emphysema through modulation of SIRT1/MMP-9 pathways.Saturated-efferocytosis generates pro-resolving CD11b low macrophages: modulation by resolvins and glucocorticoidsChronic granulomatous disease: overview and hematopoietic stem cell transplantationCurrent concepts of hyperinflammation in chronic granulomatous disease.Impaired phagocytosis of apoptotic cells by macrophages in chronic granulomatous disease is reversed by IFN-γ in a nitric oxide-dependent mannerNeutrophil clearance: when the party is over, clean-up begins.Functional and phenotypic characteristics of alternative activation induced in human monocytes by interleukin-4 or the parasitic nematode Brugia malayi.Adaptive immune defects against glycoantigens in chronic granulomatous disease via dysregulated nitric oxide productionTaurine Chloramine Stimulates Efferocytosis Through Upregulation of Nrf2-Mediated Heme Oxygenase-1 Expression in Murine Macrophages: Possible Involvement of Carbon MonoxideModulation of macrophage efferocytosis in inflammation.New Lives Given by Cell Death: Macrophage Differentiation Following Their Encounter with Apoptotic Leukocytes during the Resolution of InflammationNOX2 As a Target for Drug Development: Indications, Possible Complications, and Progress.Regulation of innate immunity by NADPH oxidase.Reactive oxygen species and thiol redox signaling in the macrophage biology of atherosclerosis.Macrophage peroxisome proliferator-activated receptor γ deficiency delays skin wound healing through impairing apoptotic cell clearance in mice.NADPH oxidase controls neutrophilic response to sterile inflammation in mice by regulating the IL-1α/G-CSF axis.VIP treatment prevents embryo resorption by modulating efferocytosis and activation profile of maternal macrophages in the CBAxDBA resorption prone modelNADPH oxidase and Nrf2 regulate gastric aspiration-induced inflammation and acute lung injury.Reactive oxygen species produced by the NADPH oxidase 2 complex in monocytes protect mice from bacterial infections.PPARγ activation following apoptotic cell instillation promotes resolution of lung inflammation and fibrosis via regulation of efferocytosis and proresolving cytokines.An efferocytosis-induced, IL-4-dependent macrophage-iNKT cell circuit suppresses sterile inflammation and is defective in murine CGDEnhanced resolution of experimental ARDS through IL-4-mediated lung macrophage reprogrammingNADPH oxidase promotes neutrophil extracellular trap formation in pulmonary aspergillosis.Hanging in the balance: endogenous anti-inflammatory mechanisms in tissue repair and fibrosis.
P2860
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P2860
Impaired apoptotic cell clearance in CGD due to altered macrophage programming is reversed by phosphatidylserine-dependent production of IL-4
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 24 October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Impaired apoptotic cell cleara ...... e-dependent production of IL-4
@en
Impaired apoptotic cell cleara ...... -dependent production of IL-4.
@nl
type
label
Impaired apoptotic cell cleara ...... e-dependent production of IL-4
@en
Impaired apoptotic cell cleara ...... -dependent production of IL-4.
@nl
prefLabel
Impaired apoptotic cell cleara ...... e-dependent production of IL-4
@en
Impaired apoptotic cell cleara ...... -dependent production of IL-4.
@nl
P2093
P2860
P1433
P1476
Impaired apoptotic cell cleara ...... e-dependent production of IL-4
@en
P2093
Brian L Harry
David W H Riches
Donna L Bratton
Kathleen McPhillips
Peter M Henson
R William Vandivier
Ruby F Fernandez-Boyanapalli
S Courtney Frasch
P2860
P304
P356
10.1182/BLOOD-2008-05-160564
P407
P577
2008-10-24T00:00:00Z