Behavioral recovery from acute hypoxia is reliant on leptin.
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Accelerated recovery from acute hypoxia in obese mice is due to obesity-associated up-regulation of interleukin-1 receptor antagonist.Sickness behavior induced by endotoxin can be mitigated by the dietary soluble fiber, pectin, through up-regulation of IL-4 and Th2 polarization.Fasting Induces IL-1 Resistance and Free-Fatty Acid-Mediated Up-Regulation of IL-1R2 and IL-1RAA narrative review on the similarities and dissimilarities between myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) and sickness behavior.Hypoxia/reoxygenation impairs memory formation via adenosine-dependent activation of caspase 1Fasting induces an anti-inflammatory effect on the neuroimmune system which a high-fat diet preventsPsychoneuroimmune implications of type 2 diabetes: reduxBiomarkers for chronic fatigue.Assessing the Functional Role of Leptin in Energy Homeostasis and the Stress Response in Vertebrates.Increased plasma interleukin-1 receptor antagonist levels in men with visceral obesity.
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Behavioral recovery from acute hypoxia is reliant on leptin.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 27 September 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Behavioral recovery from acute hypoxia is reliant on leptin.
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Behavioral recovery from acute hypoxia is reliant on leptin.
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type
label
Behavioral recovery from acute hypoxia is reliant on leptin.
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Behavioral recovery from acute hypoxia is reliant on leptin.
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prefLabel
Behavioral recovery from acute hypoxia is reliant on leptin.
@en
Behavioral recovery from acute hypoxia is reliant on leptin.
@nl
P2093
P2860
P1476
Behavioral recovery from acute hypoxia is reliant on leptin.
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P2093
Christina L Sherry
Gregory G Freund
Jason M Kramer
Jason M York
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P304
P356
10.1016/J.BBI.2008.09.011
P577
2008-09-27T00:00:00Z