MAVS regulates apoptotic cell death by decreasing K48-linked ubiquitination of voltage-dependent anion channel 1 - Wikidata - wikimedia - TriplyDB

MAVS regulates apoptotic cell death by decreasing K48-linked ubiquitination of voltage-dependent anion channel 1

MAVS regulates apoptotic cell death by decreasing K48-linked ubiquitination of voltage-dependent anion channel 1

http://www.wikidata.org/entity/Q37122923

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Innate immune pattern recognition: a cell biological perspective Lysine 63-linked TANK-binding kinase 1 ubiquitination by mindbomb E3 ubiquitin protein ligase 2 is mediated by the mitochondrial antiviral signaling protein.MAVS-MKK7-JNK2 defines a novel apoptotic signaling pathway during viral infection.Overexpression of the transcription factor Sp1 activates the OAS-RNAse L-RIG-I pathway.Amplified RLR signaling activation through an interferon-stimulated gene-endoplasmic reticulum stress-mitochondrial calcium uniporter protein loop.Novel Biomarker Proteins in Chronic Lymphocytic Leukemia: Impact on Diagnosis, Prognosis and Treatment.Curcumin inhibits the replication of enterovirus 71 in vitro IPS-1 differentially induces TRAIL, BCL2, BIRC3 and PRKCE in type I interferons-dependent and -independent anticancer activity.MAVS maintains mitochondrial homeostasis via autophagy.TAX1BP1 Restrains Virus-Induced Apoptosis by Facilitating Itch-Mediated Degradation of the Mitochondrial Adaptor MAVS.Promising Targets for Cancer Immunotherapy: TLRs, RLRs, and STING-Mediated Innate Immune Pathways MAVS Coordination of Antiviral Innate Immunity.Emerging complexity and new roles for the RIG-I-like receptors in innate antiviral immunity.MAVS-dependent host species range and pathogenicity of human hepatitis A virus.Highly Pathogenic Porcine Reproductive and Respiratory Syndrome Virus Nsp4 Cleaves VISA to Impair Antiviral Responses Mediated by RIG-I-like Receptors.Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics.Protein-protein interaction networks as a new perspective to evaluate distinct functional roles of voltage-dependent anion channel isoforms.Peroxisomes support human herpesvirus 8 latency by stabilizing the viral oncogenic protein vFLIP via the MAVS-TRAF complex.

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MAVS regulates apoptotic cell death by decreasing K48-linked ubiquitination of voltage-dependent anion channel 1

http://www.wikidata.org/entity/Q37122923

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

MAVS regulates apoptotic cell ...... tage-dependent anion channel 1

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MAVS regulates apoptotic cell ...... age-dependent anion channel 1.

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type

label

MAVS regulates apoptotic cell ...... tage-dependent anion channel 1

@en

MAVS regulates apoptotic cell ...... age-dependent anion channel 1.

@nl

prefLabel

MAVS regulates apoptotic cell ...... tage-dependent anion channel 1

@en

MAVS regulates apoptotic cell ...... age-dependent anion channel 1.

@nl

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Molecular and Cellular Biology

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MAVS regulates apoptotic cell ...... tage-dependent anion channel 1

@en

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Changzhi Xu

http://www.w3.org/2001/XMLSchema#string

Congwen Wei

http://www.w3.org/2001/XMLSchema#string

Hui Zhong

http://www.w3.org/2001/XMLSchema#string

Jia Li

http://www.w3.org/2001/XMLSchema#string

Kai Guan

http://www.w3.org/2001/XMLSchema#string

Quanbin Xu

http://www.w3.org/2001/XMLSchema#string

Shengli Ma

http://www.w3.org/2001/XMLSchema#string

Ting Song

http://www.w3.org/2001/XMLSchema#string

Xiang He

http://www.w3.org/2001/XMLSchema#string

Xiaoli Yang

http://www.w3.org/2001/XMLSchema#string

P2860

Peroxisomes are signaling platforms for antiviral innate immunity

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

Innate immune recognition of viral infection

Degradation of RIG-I following cytomegalovirus infection is independent of apoptosis

The Interferon Stimulator Mitochondrial Antiviral Signaling Protein Facilitates Cell Death by Disrupting the Mitochondrial Membrane Potential and by Activating Caspases

The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responses

Identification and characterization of MAVS, a mitochondrial antiviral signaling protein that activates NF-kappaB and IRF 3

VISA is an adapter protein required for virus-triggered IFN-beta signaling

Identification of the protein-protein contact site and interaction mode of human VDAC1 with Bcl-2 family proteins

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3137-3149

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scholarly article

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10.1128/MCB.00030-13

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16

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33

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2013-06-10T00:00:00Z

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23754752

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apoptotic process

protein ubiquitination

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3753917

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