The intracellular environment of human macrophages that produce nitric oxide promotes growth of mycobacteria.
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Innate Immune Defenses in Human Tuberculosis: An Overview of the Interactions between Mycobacterium tuberculosis and Innate Immune Cellsbis-Molybdopterin guanine dinucleotide is required for persistence of Mycobacterium tuberculosis in guinea pigs.Mycobacterium tuberculosis requires the ECF sigma factor SigE to arrest phagosome maturation.Lipid metabolism and Type VII secretion systems dominate the genome scale virulence profile of Mycobacterium tuberculosis in human dendritic cells.Mycobacterium tuberculosis Is a Natural Ornithine Aminotransferase (rocD) Mutant and Depends on Rv2323c for Growth on ArginineTrypanosoma cruzi Needs a Signal Provided by Reactive Oxygen Species to Infect MacrophagesRole of Alanine Dehydrogenase of Mycobacterium tuberculosis during Recovery from Hypoxic Nonreplicating Persistence.Game of 'Somes: Protein Destruction for Mycobacterium tuberculosis Pathogenesis.Nitrate, nitrite and nitric oxide reductases: from the last universal common ancestor to modern bacterial pathogens.Phosphocholine-containing ligands direct CRP induction of M2 macrophage polarization independent of T cell polarization: Implication for chronic inflammatory states.Nitrite reductase NirBD is induced and plays an important role during in vitro dormancy of Mycobacterium tuberculosisNitrite produced by Mycobacterium tuberculosis in human macrophages in physiologic oxygen impacts bacterial ATP consumption and gene expression.Epigenetic silencing of the human NOS2 gene: rethinking the role of nitric oxide in human macrophage inflammatory responses.Mycobacterial Dormancy Systems and Host Responses in Tuberculosis.Endogenous and Exogenous KdpF Peptide Increases Susceptibility of Mycobacterium bovis BCG to Nitrosative Stress and Reduces Intramacrophage Replication.In search of a new paradigm for protective immunity to TBNitrogen metabolism in Mycobacterium tuberculosis physiology and virulence.Phylogenomics of Mycobacterium Nitrate Reductase Operon.Oxidative Phosphorylation as a Target Space for Tuberculosis: Success, Caution, and Future Directions.Comparative evaluation of in vitro human macrophage models for mycobacterial infection study.IL-12 and IL-27 regulate the phagolysosomal pathway in mycobacteria-infected human macrophages.STAT3 Represses Nitric Oxide Synthesis in Human Macrophages upon Mycobacterium tuberculosis Infection.Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts.Nitric Oxide in the Pathogenesis and Treatment of Tuberculosis.Priming of innate antimycobacterial immunity by heat-killed Listeria monocytogenes induces sterilizing response in the adult zebrafish tuberculosis model.Regulation of Three Virulence Strategies of Mycobacterium tuberculosis: A Success Story.Chlorate Specifically Targets Oxidant-Starved, Antibiotic-Tolerant Populations of Pseudomonas aeruginosa Biofilms
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The intracellular environment of human macrophages that produce nitric oxide promotes growth of mycobacteria.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 17 June 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The intracellular environment ...... omotes growth of mycobacteria.
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The intracellular environment ...... omotes growth of mycobacteria.
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type
label
The intracellular environment ...... omotes growth of mycobacteria.
@en
The intracellular environment ...... omotes growth of mycobacteria.
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prefLabel
The intracellular environment ...... omotes growth of mycobacteria.
@en
The intracellular environment ...... omotes growth of mycobacteria.
@nl
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P2860
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The intracellular environment ...... romotes growth of mycobacteria
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Charles D Sohaskey
Cory M Robinson
Franz-Christoph Bange
Joo-Yong Jung
Jyothi Rengarajan
P2860
P304
P356
10.1128/IAI.00611-13
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P577
2013-06-17T00:00:00Z