Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis. - Wikidata - wikimedia - TriplyDB

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

http://www.wikidata.org/entity/Q37125776

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Loss of Grainy head-like 1 is associated with disruption of the epidermal barrier and squamous cell carcinoma of the skin Macrophages and fibroblasts during inflammation and tissue repair in models of organ regeneration.Loss of epidermal p38α signaling prevents UVR-induced inflammation via acute and chronic mechanisms.Updating the Wnt pathways.The danger model approach to the pathogenesis of the rheumatic diseases.Mesenchymal Wnt signaling promotes formation of sternum and thoracic body wall Wnts are dispensable for differentiation and self-renewal of adult murine hematopoietic stem cells.Axin2 marks quiescent hair follicle bulge stem cells that are maintained by autocrine Wnt/β-catenin signaling.Epidermal Nbn deletion causes premature hair loss and a phenotype resembling psoriasiform dermatitis.Wnt signaling in skin homeostasis and pathology.Distinct functions for Wnt/β-catenin in hair follicle stem cell proliferation and survival and interfollicular epidermal homeostasis.Wnt Signalling Pathways in Skin Development and Epidermal Stem Cells.Transcription Factor CTIP1/ BCL11A Regulates Epidermal Differentiation and Lipid Metabolism During Skin Development.Angiocrine Wnt signaling controls liver growth and metabolic maturation in mice.Phospholipase Cδ1 regulates p38 MAPK activity and skin barrier integrity.DLX3-dependent STAT3 signaling in keratinocytes regulates skin immune homeostasis.Intraindividual genome expression analysis reveals a specific molecular signature of psoriasis and eczema.Tubule-Derived Wnts Are Required for Fibroblast Activation and Kidney Fibrosis.Epigenetic downregulation of SFRP4 contributes to epidermal hyperplasia in psoriasis.

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Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

http://www.wikidata.org/entity/Q37125776

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 05 August 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@en

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@nl

type

label

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@en

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@nl

prefLabel

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@en

Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis.

@nl

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Journal of Experimental Medicine

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Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis

@en

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Claudia Korn

http://www.w3.org/2001/XMLSchema#string

Cornelia Mauch

http://www.w3.org/2001/XMLSchema#string

Daniel Baumann

http://www.w3.org/2001/XMLSchema#string

Grainne Kerr

http://www.w3.org/2001/XMLSchema#string

Iris Augustin

http://www.w3.org/2001/XMLSchema#string

Julia Gross

http://www.w3.org/2001/XMLSchema#string

Tamara Grigoryan

http://www.w3.org/2001/XMLSchema#string

Walter Birchmeier

http://www.w3.org/2001/XMLSchema#string

P2860

Adenomatous polyposis coli (APC) is required for normal development of skin and thymus

Wnt5a exhibits layer-specific expression in adult skin, is upregulated in psoriasis, and synergizes with type 1 interferon

Wntless, a conserved membrane protein dedicated to the secretion of Wnt proteins from signaling cells

Secretion of Wnt ligands requires Evi, a conserved transmembrane protein

Psoriasis genetics: breaking the barrier

Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation

Wnt/β-catenin signaling and disease

S100A9/S100A8: Myeloid representatives of the S100 protein family as prominent players in innate immunity

beta-Catenin controls hair follicle morphogenesis and stem cell differentiation in the skin

The skin: an indispensable barrier

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1761-1777

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scholarly article

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10.1084/JEM.20121871

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9

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210

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2013-08-05T00:00:00Z

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255688306

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23918954

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3754868

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