The HIV-1 protein Vpr targets the endoribonuclease Dicer for proteasomal degradation to boost macrophage infection
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Visualizing Vpr-induced G2 arrest and apoptosisCullin4A and cullin4B are interchangeable for HIV Vpr and Vpx action through the CRL4 ubiquitin ligase complexHIV life cycle, innate immunity and autophagy in the central nervous systemHIV-1 Vpr induces the degradation of ZIP and sZIP, adaptors of the NuRD chromatin remodeling complex, by hijacking DCAF1/VprBP.The many faces of Dicer: the complexity of the mechanisms regulating Dicer gene expression and enzyme activitiesHIV-1 Vpr Protein Enhances Proteasomal Degradation of MCM10 DNA Replication Factor through the Cul4-DDB1[VprBP] E3 Ubiquitin Ligase to Induce G2/M Cell Cycle Arrest.RNA Viruses: RNA Roles in Pathogenesis, Coreplication and Viral LoadDysfunctional Subcutaneous Fat With Reduced Dicer and Brown Adipose Tissue Gene Expression in HIV-Infected Patients.HIV-1 Vpr degrades the HLTF DNA translocase in T cells and macrophages.Robust RNAi enhancement via human Argonaute-2 overexpression from plasmids, viral vectors and cell lines.CUL4A ubiquitin ligase: a promising drug target for cancer and other human diseasesHIV-1 Vpr-a still "enigmatic multitasker".The ubiquitin-conjugating system: multiple roles in viral replication and infection.HIV-1 Vpr Protein Induces Proteasomal Degradation of Chromatin-associated Class I HDACs to Overcome Latent Infection of Macrophages.MicroRNA regulation of viral immunity, latency, and carcinogenesis of selected tumor viruses and HIV.HIV1-viral protein R (Vpr) mutations: associated phenotypes and relevance for clinical pathologies.Vpr Promotes Macrophage-Dependent HIV-1 Infection of CD4+ T Lymphocytes.Changes in the cellular microRNA profile by the intracellular expression of HIV-1 Tat regulator: A potential mechanism for resistance to apoptosis and impaired proliferation in HIV-1 infected CD4+ T cells.Hijacking of the Ubiquitin/Proteasome Pathway by the HIV Auxiliary Proteins.DCAF1 is involved in HCV replication through regulation of miR-122.Multiple Inhibitory Factors Act in the Late Phase of HIV-1 Replication: a Systematic Review of the Literature.JNK-mediated microglial DICER degradation potentiates inflammatory responses to induce dopaminergic neuron loss.
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The HIV-1 protein Vpr targets the endoribonuclease Dicer for proteasomal degradation to boost macrophage infection
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 July 2013
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vedecký článok
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videnskabelig artikel
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The HIV-1 protein Vpr targets ...... to boost macrophage infection
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The HIV-1 protein Vpr targets ...... to boost macrophage infection.
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The HIV-1 protein Vpr targets ...... to boost macrophage infection
@en
The HIV-1 protein Vpr targets ...... to boost macrophage infection.
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The HIV-1 protein Vpr targets ...... to boost macrophage infection
@en
The HIV-1 protein Vpr targets ...... to boost macrophage infection.
@nl
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The HIV-1 protein Vpr targets ...... to boost macrophage infection
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Andrea K M Furuya
Carlos M C de Noronha
Hamayun J Sharifi
Laurieann Casey Klockow
Michael Nekorchuk
Xiaoyun Wen
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P304
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10.1016/J.VIROL.2013.06.010
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P577
2013-07-09T00:00:00Z