MARCKS regulates growth and radiation sensitivity and is a novel prognostic factor for glioma. - Wikidata - wikimedia - TriplyDB

MARCKS regulates growth and radiation sensitivity and is a novel prognostic factor for glioma.

MARCKS regulates growth and radiation sensitivity and is a novel prognostic factor for glioma.

http://www.wikidata.org/entity/Q37130116

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X MARCKS the spot: myristoylated alanine-rich C kinase substrate in neuronal function and disease MARCKS phosphorylation is modulated by a peptide mimetic of MARCKS effector domain leading to increased radiation sensitivity in lung cancer cell lines Kinomic exploration of temozolomide and radiation resistance in Glioblastoma multiforme xenolines.Global characterization of signalling networks associated with tamoxifen resistance in breast cancer.The Effector Domain of MARCKS Is a Nuclear Localization Signal that Regulates Cellular PIP2 Levels and Nuclear PIP2 Localization.MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27kip1- Dependent Mechanism.A peptide that inhibits function of Myristoylated Alanine-Rich C Kinase Substrate (MARCKS) reduces lung cancer metastasis.Network Modeling Identifies Patient-specific Pathways in Glioblastoma.MARCKS contributes to stromal cancer-associated fibroblast activation and facilitates ovarian cancer metastasis.Targeting phospho-MARCKS overcomes drug-resistance and induces antitumor activity in preclinical models of multiple myeloma.Upregulation of MARCKS in kidney cancer and its potential as a therapeutic target.Functional characterization of the tumor-suppressor MARCKS in colorectal cancer and its association with survival.MARCKS promotes invasion and is associated with biochemical recurrence in prostate cancer.A novel predictor of cancer malignancy: up-regulation of myristoylated alanine-rich C kinase substrate phosphorylation in lung cancer.Myristoylated alanine-rich C kinase substrate (MARCKS): a multirole signaling protein in cancers.Carnosic acid protects mice from high-fat diet-induced NAFLD by regulating MARCKS.Combinatorial Drug Testing in 3D Microtumors Derived from GBM Patient-Derived Xenografts Reveals Cytotoxic Synergy in Pharmacokinomics-informed Pathway Interactions.Kinomics toolbox-A web platform for analysis and viewing of kinomic peptide array data Overexpression of MARCKS indicates a poor prognosis of oral squamous cell carcinoma

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MARCKS regulates growth and radiation sensitivity and is a novel prognostic factor for glioma.

http://www.wikidata.org/entity/Q37130116

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 22 May 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

MARCKS regulates growth and ra ...... prognostic factor for glioma.

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MARCKS regulates growth and ra ...... prognostic factor for glioma.

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type

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MARCKS regulates growth and ra ...... prognostic factor for glioma.

@en

MARCKS regulates growth and ra ...... prognostic factor for glioma.

@nl

prefLabel

MARCKS regulates growth and ra ...... prognostic factor for glioma.

@en

MARCKS regulates growth and ra ...... prognostic factor for glioma.

@nl

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1078-0432.CCR-11-3091

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Clinical Cancer Research

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MARCKS regulates growth and ra ...... prognostic factor for glioma.

@en

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Alexander C Whitley

http://www.w3.org/2001/XMLSchema#string

Christine W Duarte

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Christopher D Willey

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Eddy S Yang

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G Yancey Gillespie

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James A Bonner

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John S Jarboe

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Joshua C Anderson

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Patricia H Hicks

http://www.w3.org/2001/XMLSchema#string

Raymond O McCubrey

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P2860

MARCKS is an actin filament crosslinking protein regulated by protein kinase C and calcium-calmodulin

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Exciting new advances in neuro-oncology: the avenue to a cure for malignant glioma

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Molecular genetic analysis of giant cell glioblastomas

MGMT gene silencing and benefit from temozolomide in glioblastoma.

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

Myristoylated, alanine-rich C-kinase substrate phosphorylation regulates growth cone adhesion and pathfinding

Effects of radiotherapy with concomitant and adjuvant temozolomide versus radiotherapy alone on survival in glioblastoma in a randomised phase III study: 5-year analysis of the EORTC-NCIC trial

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3030-3041

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scholarly article

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10.1158/1078-0432.CCR-11-3091

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11

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2012-05-22T00:00:00Z

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225042129

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3756473

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