Suppression of radiation-induced DNA double-strand break repair by MyD88 is accompanied by apoptosis and crypt loss in mouse colon. - Wikidata - wikimedia - TriplyDB

Suppression of radiation-induced DNA double-strand break repair by MyD88 is accompanied by apoptosis and crypt loss in mouse colon.

Suppression of radiation-induced DNA double-strand break repair by MyD88 is accompanied by apoptosis and crypt loss in mouse colon.

http://www.wikidata.org/entity/Q37137257

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TLR4/PKC-mediated tight junction modulation: a clinical marker of chemotherapy-induced gut toxicity?Pattern recognition receptors and DNA repair: starting to put a jigsaw puzzle together Radiation takes its Toll Blockade of TLR3 protects mice from lethal radiation-induced gastrointestinal syndrome.Cancer-associated toll-like receptor modulation and insinuation in infection susceptibility: association or coincidence?The intestinal microbiota in allogeneic hematopoietic cell transplant and graft-versus-host disease.Sam68/KHDRBS1-dependent NF-κB activation confers radioprotection to the colon epithelium in γ-irradiated mice.Hydrogen-water ameliorates radiation-induced gastrointestinal toxicity via MyD88's effects on the gut microbiota.

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Suppression of radiation-induced DNA double-strand break repair by MyD88 is accompanied by apoptosis and crypt loss in mouse colon.

http://www.wikidata.org/entity/Q37137257

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article científic

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 12 August 2013

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vedecký článok

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vetenskaplig artikel

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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Suppression of radiation-induc ...... and crypt loss in mouse colon.

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L J Egan

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The apoptotic signaling pathway activated by Toll-like receptor-2

Megabase chromatin domains involved in DNA double-strand breaks in vivo

Spatial organization of the mammalian genome surveillance machinery in response to DNA strand breaks

Prostaglandin E2 reduces radiation-induced epithelial apoptosis through a mechanism involving AKT activation and bax translocation

Regulation of spontaneous intestinal tumorigenesis through the adaptor protein MyD88

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Toll-like receptor 4-dependent contribution of the immune system to anticancer chemotherapy and radiotherapy

Nuclear foci of mammalian Rad51 recombination protein in somatic cells after DNA damage and its localization in synaptonemal complexes

Toll-like receptor-4 promotes the development of colitis-associated colorectal tumors.

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10.1038/ONCSIS.2013.22

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apoptotic process

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