Contribution of the sympathetic hormone epinephrine to the sensitizing effect of ethanol on LPS-induced liver damage in mice.

Contribution of the sympathetic hormone epinephrine to the sensitizing effect of ethanol on LPS-induced liver damage in mice.

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http://www.wikidata.org/entity/Q37139288
Defining hepatic dysfunction parameters in two models of fatty liver disease in zebrafish larvaeCatecholamine stress alters neutrophil trafficking and impairs wound healing by β2-adrenergic receptor-mediated upregulation of IL-6.PAI-1 plays a protective role in CCl4-induced hepatic fibrosis in mice: role of hepatocyte division."Second hit" models of alcoholic liver disease.Ethanol and liver: recent insights into the mechanisms of ethanol-induced fatty liverToll-like receptor-mediated signaling cascade as a regulator of the inflammation network during alcoholic liver disease.Staphylococcus aureus transporters Hts, Sir, and Sst capture iron liberated from human transferrin by Staphyloferrin A, Staphyloferrin B, and catecholamine stress hormones, respectively, and contribute to virulence.Xiaochaihu Decoction attenuates the vicious circle between the oxidative stress and the ALP inactivation through LPS-catecholamines interactions in gut, liver and brain during CCI4+ethanol-induced mouse HCC.The role of alpha-1 and alpha-2 adrenoceptors in restraint stress-induced liver injury in mice.Vinyl Chloride Metabolites Potentiate Inflammatory Liver Injury Caused by LPS in Mice.Alcohol metabolites and lipopolysaccharide: roles in the development and/or progression of alcoholic liver disease.Role of innate immunity and the microbiota in liver fibrosis: crosstalk between the liver and gut.Toll-like receptors in alcoholic liver disease, non-alcoholic steatohepatitis and carcinogenesis.Chronic Alcohol Exposure Enhances Lipopolysaccharide-Induced Lung Injury in Mice: Potential Role of Systemic Tumor Necrosis Factor-AlphaFibrin accumulation plays a critical role in the sensitization to lipopolysaccharide-induced liver injury caused by ethanol in mice.Plasminogen Activator Inhibitor-1 Is Critical in Alcohol-Enhanced Acute Lung Injury in Mice.Carvedilol improves ethanol-induced liver injury via modifying the interaction between oxidative stress and sympathetic hyperactivity in rats.
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Contribution of the sympathetic hormone epinephrine to the sensitizing effect of ethanol on LPS-induced liver damage in mice.

Item
http://www.wikidata.org/entity/Q37139288
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 March 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Contribution of the sympatheti ...... -induced liver damage in mice.
@en
Contribution of the sympatheti ...... -induced liver damage in mice.
@nl
type
label
Contribution of the sympatheti ...... -induced liver damage in mice.
@en
Contribution of the sympatheti ...... -induced liver damage in mice.
@nl
prefLabel
Contribution of the sympatheti ...... -induced liver damage in mice.
@en
Contribution of the sympatheti ...... -induced liver damage in mice.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P921
P932
P356
P1433
P1476
Contribution of the sympatheti ...... -induced liver damage in mice.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P921
P932