Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart - Wikidata - wikimedia - TriplyDB

Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart

Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart

http://www.wikidata.org/entity/Q37143342

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Ca(2+) signaling in the myocardium by (redox) regulation of PKA/CaMKII CaMKII oxidative activation and the pathogenesis of cardiac disease Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias New therapeutic targets in cardiology: arrhythmias and Ca2+/calmodulin-dependent kinase II (CaMKII)Protein methionine oxidation augments reperfusion injury in acute ischemic stroke Overexpression of CARMA3 in non-small-cell lung cancer is linked for tumor progression CaMKII determines mitochondrial stress responses in heart Activation of complement factor B contributes to murine and human myocardial ischemia/reperfusion injury.Three ways to die suddenly: do they all require calcium calmodulin-dependent protein kinase II?Inhibition of CaMKII does not attenuate cardiac hypertrophy in mice with dysfunctional ryanodine receptor Pivotal advance: The pattern recognition receptor ligands lipopolysaccharide and polyinosine-polycytidylic acid stimulate factor B synthesis by the macrophage through distinct but overlapping mechanisms The multifunctional Ca2+/calmodulin-dependent kinase II delta (CaMKIIdelta) controls neointima formation after carotid ligation and vascular smooth muscle cell proliferation through cell cycle regulation by p21.CARMA3: A novel scaffold protein in regulation of NF-κB activation and diseases Complement dependency of cardiomyocyte release of mediators during sepsis CaMKII is essential for the proasthmatic effects of oxidation.Oxidized CaMKII causes cardiac sinus node dysfunction in mice.Progress toward the prevention and treatment of atrial fibrillation: A summary of the Heart Rhythm Society Research Forum on the Treatment and Prevention of Atrial Fibrillation, Washington, DC, December 9-10, 2013.Glycoproteins identified from heart failure and treatment models MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarction.Oxidation of CaMKII determines the cardiotoxic effects of aldosterone.Dual Activation of TRIF and MyD88 Adaptor Proteins by Angiotensin II Evokes Opposing Effects on Pressure, Cardiac Hypertrophy, and Inflammatory Gene Expression.The multifunctional Ca2+/calmodulin-dependent kinase II regulates vascular smooth muscle migration through matrix metalloproteinase 9.Mammalian DNA is an endogenous danger signal that stimulates local synthesis and release of complement factor B.Intraductal injection of LPS as a mouse model of mastitis: signaling visualized via an NF-κB reporter transgenic.CaMKII inhibition in type II pneumocytes protects from bleomycin-induced pulmonary fibrosis by preventing Ca2+-dependent apoptosis.Atrial remodelling in atrial fibrillation: CaMKII as a nodal proarrhythmic signal Interaction of β1-adrenoceptor with RAGE mediates cardiomyopathy via CaMKII signaling.Endothelial PPAR-γ provides vascular protection from IL-1β-induced oxidative stress Is CaMKII a link between inflammation and hypertrophy in heart?Cardiomyocyte-specific p65 NF-κB deletion protects the injured heart by preservation of calcium handling.Oxidant stress promotes disease by activating CaMKII.Complement C3c as a biomarker in heart failure.Complement factor B is the downstream effector of TLRs and plays an important role in a mouse model of severe sepsis.Oxidized CaMKII promotes asthma through the activation of mast cells.Mitochondrial CaMKII inhibition in airway epithelium protects against allergic asthma.CaMKII in myocardial hypertrophy and heart failure CaMKII in the cardiovascular system: sensing redox states.The promise of CaMKII inhibition for heart disease: preventing heart failure and arrhythmias.CaMKII inhibitors: from research tools to therapeutic agents Integrated mechanisms of CaMKII-dependent ventricular remodeling.

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Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart

http://www.wikidata.org/entity/Q37143342

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 March 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Ca2+/calmodulin-dependent kina ...... expression in the mouse heart

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Ca2+/calmodulin-dependent kina ...... expression in the mouse heart.

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Ca2+/calmodulin-dependent kina ...... expression in the mouse heart

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Ca2+/calmodulin-dependent kina ...... expression in the mouse heart.

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Ca2+/calmodulin-dependent kina ...... expression in the mouse heart

@en

Ca2+/calmodulin-dependent kina ...... expression in the mouse heart.

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Journal of Clinical Investigation

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Ca2+/calmodulin-dependent kina ...... expression in the mouse heart

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Ann Kapoun

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Fiona E Yull

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Jinying Yang

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John S Lowe

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Joshua M Thurman

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Kathy Zimmermann

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Linda Higgins

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Madhu V Singh

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Mark E Anderson

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Minati Singh

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P2860

CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

Ca2+/calmodulin-dependent protein kinase II is a modulator of CARMA1-mediated NF-kappaB activation

Targeted cardiac overexpression of A20 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction

Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart

Characterization of a calmodulin kinase II inhibitor protein in brain

Within the fold: assessing differential expression measures and reproducibility in microarray assays.

Toll-like receptors

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

NF-kappa B functions in synaptic signaling and behavior

Biomarkers in heart failure

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10.1172/JCI35814

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4

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19273909

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