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Early biomarkers and potential mediators of ventilation-induced lung injury in very preterm lambs

Early biomarkers and potential mediators of ventilation-induced lung injury in very preterm lambs

http://www.wikidata.org/entity/Q37144027

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The immune consequences of preterm birth Positive end-expiratory pressure and surfactant decrease lung injury during initiation of ventilation in fetal sheep Cardiorespiratory Monitoring during Neonatal Resuscitation for Direct Feedback and Audit Unraveling the Links Between the Initiation of Ventilation and Brain Injury in Preterm Infants Protective efficacy of orally administered, heat-killed Lactobacillus pentosus b240 against influenza A virus.Antenatal and postnatal corticosteroid and resuscitation induced lung injury in preterm sheep.Surfactant protein-d inhibits lung inflammation caused by ventilation in premature newborn lambs Inhibitors of inflammation and endogenous surfactant pool size as modulators of lung injury with initiation of ventilation in preterm sheep.High bias gas flows increase lung injury in the ventilated preterm lamb.Protective ventilation of preterm lambs exposed to acute chorioamnionitis does not reduce ventilation-induced lung or brain injury.Inflammation and lung maturation from stretch injury in preterm fetal sheep.CTGF disrupts alveolarization and induces pulmonary hypertension in neonatal mice: implication in the pathogenesis of severe bronchopulmonary dysplasia Mechanical ventilation injury and repair in extremely and very preterm lungs.Targeting inflammation to prevent bronchopulmonary dysplasia: can new insights be translated into therapies?Pressure- versus volume-limited sustained inflations at resuscitation of premature newborn lambs.CYR61 (CCN1) overexpression induces lung injury in mice Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasia Pressure-limited sustained inflation vs. gradual tidal inflations for resuscitation in preterm lambs.Tracheal Aspirate Levels of the Matricellular Protein SPARC Predict Development of Bronchopulmonary Dysplasia Interfacial stress affects rat alveolar type II cell signaling and gene expression.Human amnion epithelial cells modulate the inflammatory response to ventilation in preterm lambs.IL-1β processing in mechanical ventilation-induced inflammation is dependent on neutrophil factors rather than caspase-1 Mechanisms of bronchopulmonary dysplasia.Selection of Reference Genes for Gene Expression Studies related to lung injury in a preterm lamb model.Platelet-derived growth factor-A regulates lung fibroblast S-phase entry through p27(kip1) and FoxO3a Lucinactant attenuates pulmonary inflammatory response, preserves lung structure, and improves physiologic outcomes in a preterm lamb model of RDS Airway injury from initiating ventilation in preterm sheep.Application of continuous positive airway pressure in the delivery room: a multicenter randomized clinical trial.Ventilator-induced lung injury in preterm infants.Bronchopulmonary dysplasia - an overview about pathophysiologic concepts.Does growth restriction increase the vulnerability to acute ventilation-induced brain injury in newborn lambs? Implications for future health and disease.Lipopolysaccharide-induced CCN1 production enhances interleukin-6 secretion in bronchial epithelial cells.Surfactant instillation in spontaneously breathing preterm infants: a systematic review and meta-analysis.Delivery room interventions to prevent bronchopulmonary dysplasia in extremely preterm infants.A comparison of high-frequency jet ventilation and synchronised intermittent mandatory ventilation in preterm lambs.Glycogen synthase kinase-3β/β-catenin signaling regulates neonatal lung mesenchymal stromal cell myofibroblastic differentiation.Establishing lung gas volumes at birth: interaction between positive end-expiratory pressures and tidal volumes in preterm rabbits.Surfactant before the first inflation at birth improves spatial distribution of ventilation and reduces lung injury in preterm lambs.Sustained versus intermittent lung inflation for resuscitation of preterm infants: a randomized controlled trial.Lung fluid absorption is induced in preterm guinea pigs ventilated with low tidal volumes.

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Early biomarkers and potential mediators of ventilation-induced lung injury in very preterm lambs

http://www.wikidata.org/entity/Q37144027

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 10 March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Early biomarkers and potential ...... g injury in very preterm lambs

@en

Early biomarkers and potential ...... injury in very preterm lambs.

@nl

type

label

Early biomarkers and potential ...... g injury in very preterm lambs

@en

Early biomarkers and potential ...... injury in very preterm lambs.

@nl

prefLabel

Early biomarkers and potential ...... g injury in very preterm lambs

@en

Early biomarkers and potential ...... injury in very preterm lambs.

@nl

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1465-9921-10-19

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Respiratory Research

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Early biomarkers and potential ...... g injury in very preterm lambs

@en

P2093

Kelly Crossley

http://www.w3.org/2001/XMLSchema#string

Megan E Probyn

http://www.w3.org/2001/XMLSchema#string

Peter G Davis

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Stuart B Hooper

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Timothy J Cole

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Valerie A Zahra

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P2860

Pathways of Egr-1-mediated gene transcription in vascular biology

Luteinizing hormone deficiency and female infertility in mice lacking the transcription factor NGFI-A (Egr-1)

Egr-1, a master switch coordinating upregulation of divergent gene families underlying ischemic stress

Pulmonary hypoplasia in the connective tissue growth factor (Ctgf) null mouse

Brief, large tidal volume ventilation initiates lung injury and a systemic response in fetal sheep.

Early changes in lung gene expression due to high tidal volume.

The connective tissue growth factor/cysteine-rich 61/nephroblastoma overexpressed (CCN) family.

Induction and function of CYR61 (CCN1) in prostatic stromal and epithelial cells: CYR61 is required for prostatic cell proliferation.

Mechanisms of ventilator-induced lung injury.

Pathology of new bronchopulmonary dysplasia.

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1465-9921-10-19

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10.1186/1465-9921-10-19

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10

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Colin J. Morley

Megan J Wallace

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2009-03-10T00:00:00Z

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24199230

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