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MMP9 and SCF protect from apoptosis in acute kidney injury

MMP9 and SCF protect from apoptosis in acute kidney injury

http://www.wikidata.org/entity/Q37146471

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Matrix Metalloproteinases in Non-Neoplastic Disorders.Pathophysiological role of different tubular epithelial cell death modes in acute kidney injury Matrix metalloproteinases in kidney homeostasis and diseases Proteomic candidate biomarkers of drug-induced nephrotoxicity in the rat Hydroxyfasudil-mediated inhibition of ROCK1 and ROCK2 improves kidney function in rat renal acute ischemia-reperfusion injury The restrained expression of NF-kB in renal tissue ameliorates folic acid induced acute kidney injury in mice Stem cell factor expression after renal ischemia promotes tubular epithelial survival.c-Kit modifies the inflammatory status of smooth muscle cells c-Kit signaling determines neointimal hyperplasia in arteriovenous fistulae Increased expression of intranuclear matrix metalloproteinase 9 in atrophic renal tubules is associated with renal fibrosis.Akt1-mediated fast/glycolytic skeletal muscle growth attenuates renal damage in experimental kidney disease.Numb protects renal proximal tubular cells from puromycin aminonucleoside-induced apoptosis through inhibiting Notch signaling pathway.MMP-9 gene deletion mitigates microvascular loss in a model of ischemic acute kidney injury.c-Kit immunoexpression delineates a putative endothelial progenitor cell population in developing human lungs.Loss of matrix metalloproteinase-8 is associated with worsened recovery after ischemic kidney injury Tubule-specific ablation of endogenous β-catenin aggravates acute kidney injury in mice Unique Transcriptional Programs Identify Subtypes of AKI.Stem cell conditioned medium improves acute lung injury in mice: in vivo evidence for stem cell paracrine action.Stem cell factor improves lung recovery in rats following neonatal hyperoxia-induced lung injury.Tankyrase inhibition aggravates kidney injury in the absence of CD2AP.Bone-derived mesenchymal stromal cells from HIV transgenic mice exhibit altered proliferation, differentiation capacity and paracrine functions along with impaired therapeutic potential in kidney injury.MMP9 limits apoptosis and stimulates branching morphogenesis during kidney development.A modular microfluidic bioreactor with improved throughput for evaluation of polarized renal epithelial cells.Progress in the development of animal models of acute kidney injury and its impact on drug discovery.Use of Xenopus Frogs to Study Renal Development/Repair.Impaired response of the bronchial epithelium to inflammation characterizes severe equine asthma.Transplantation of bone marrow-derived MSCs improves cisplatinum-induced renal injury through paracrine mechanisms.Transcription profile of genes affected in response to pathological changes in drug-induced rat model of acute kidney injury.

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MMP9 and SCF protect from apoptosis in acute kidney injury

http://www.wikidata.org/entity/Q37146471

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on April 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

MMP9 and SCF protect from apoptosis in acute kidney injury

@en

MMP9 and SCF protect from apoptosis in acute kidney injury.

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type

label

MMP9 and SCF protect from apoptosis in acute kidney injury

@en

MMP9 and SCF protect from apoptosis in acute kidney injury.

@nl

prefLabel

MMP9 and SCF protect from apoptosis in acute kidney injury

@en

MMP9 and SCF protect from apoptosis in acute kidney injury.

@nl

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Journal of the American Society of Nephrology

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MMP9 and SCF protect from apoptosis in acute kidney injury

@en

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Brigitte Lelongt

http://www.w3.org/2001/XMLSchema#string

Catherine Arnould

http://www.w3.org/2001/XMLSchema#string

Emmanuel Letavernier

http://www.w3.org/2001/XMLSchema#string

Hélène Martinan de Préneuf

http://www.w3.org/2001/XMLSchema#string

Matthieu Monge

http://www.w3.org/2001/XMLSchema#string

Pierre Ronco

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Soraya Bengatta

http://www.w3.org/2001/XMLSchema#string

Zena Werb

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P2860

Development, migration, and survival of mast cells

MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes

Recruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligand

Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9)

Minocycline up-regulates Bcl-2 and protects against cell death in mitochondria

c-kit delineates a distinct domain of progenitors in the developing kidney

Expression of the type IV collagenase system during mouse kidney development and tubule segmentation.

Pathophysiology of acute renal failure.

Apoptosis and extracellular matrix-cell interactions in kidney disease.

Suppression of ICE and apoptosis in mammary epithelial cells by extracellular matrix.

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787-797

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scholarly article

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10.1681/ASN.2008050515

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4

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20

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2009-04-01T00:00:00Z

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19329763

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apoptotic process

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2663840

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