STAT5 is a key transcription factor for IL-3-mediated inhibition of RANKL-induced osteoclastogenesis
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A Conditional Knockout Mouse Model Reveals a Critical Role of PKD1 in Osteoblast Differentiation and Bone Development.STAT5-Interacting Proteins: A Synopsis of Proteins that Regulate STAT5 Activity.Interleukin-7 Induces Osteoclast Formation via STAT5, Independent of Receptor Activator of NF-kappaB Ligand.Increased Ca2+ signaling through CaV1.2 promotes bone formation and prevents estrogen deficiency-induced bone loss.Regulation of Osteoclast Differentiation by Cytokine Networks.
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STAT5 is a key transcription factor for IL-3-mediated inhibition of RANKL-induced osteoclastogenesis
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 August 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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STAT5 is a key transcription f ...... NKL-induced osteoclastogenesis
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STAT5 is a key transcription f ...... KL-induced osteoclastogenesis.
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type
label
STAT5 is a key transcription f ...... NKL-induced osteoclastogenesis
@en
STAT5 is a key transcription f ...... KL-induced osteoclastogenesis.
@nl
prefLabel
STAT5 is a key transcription f ...... NKL-induced osteoclastogenesis
@en
STAT5 is a key transcription f ...... KL-induced osteoclastogenesis.
@nl
P2093
P2860
P356
P1433
P1476
STAT5 is a key transcription f ...... NKL-induced osteoclastogenesis
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P2093
Byung-Chul Jeong
Inyoung Kim
Jongwon Lee
Jung Ha Kim
Kabsun Kim
Kyung Keun Kim
Nacksung Kim
Semun Seong
P2860
P2888
P356
10.1038/SREP30977
P407
P577
2016-08-03T00:00:00Z