Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23. - Wikidata - wikimedia - TriplyDB

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

http://www.wikidata.org/entity/Q37152270

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The role of fibroblast growth factor 23 and Klotho in uremic cardiomyopathy.FGF23 promotes myocardial fibrosis in mice through activation of β-catenin.Vitamin D treatment attenuates cardiac FGF23/FGFR4 signaling and hypertrophy in uremic rats.Cardiac actions of fibroblast growth factor 23.Mineral metabolism and cardiovascular disease in CKD.Fibroblast growth factor 23 and heart failure: the plot thickens.Genetic Ablation of Fgf23 or Klotho Does not Modulate Experimental Heart Hypertrophy Induced by Pressure Overload.Role of Vitamin D in Cardiovascular Diseases.Pleiotropic Actions of FGF23.Paracrine Effects of FGF23 on the Heart.Cardiotoxicity of Uremic Toxins: A Driver of Cardiorenal Syndrome

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Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

http://www.wikidata.org/entity/Q37152270

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 09 April 2015

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@en

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@nl

type

label

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@en

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@nl

prefLabel

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@en

Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@nl

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Journal of Bone and Mineral Research

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Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23.

@en

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Kathrin I Odörfer

http://www.w3.org/2001/XMLSchema#string

Olena Andrukhova

http://www.w3.org/2001/XMLSchema#string

Reinhold G Erben

http://www.w3.org/2001/XMLSchema#string

Svetlana Slavic

http://www.w3.org/2001/XMLSchema#string

P2860

Systematic Review: Vitamin D and Cardiometabolic Outcomes

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

Regulation and function of the FGF23/klotho endocrine pathways

Klotho converts canonical FGF receptor into a specific receptor for FGF23

Klotho lacks a vitamin D independent physiological role in glucose homeostasis, bone turnover, and steady-state PTH secretion in vivo

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

Circulating FGF-23 is regulated by 1alpha,25-dihydroxyvitamin D3 and phosphorus in vivo

Role of endogenous bone marrow cells in long-term repair mechanisms after myocardial infarction.

Vitamin D is a regulator of endothelial nitric oxide synthase and arterial stiffness in mice

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1831-1839

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scholarly article

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10.1002/JBMR.2527

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10

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30

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2015-04-09T00:00:00Z

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25858796

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