Morphine hyperalgesia gated through microglia-mediated disruption of neuronal Cl⁻ homeostasis. - Wikidata - wikimedia - TriplyDB

Morphine hyperalgesia gated through microglia-mediated disruption of neuronal Cl⁻ homeostasis.

Morphine hyperalgesia gated through microglia-mediated disruption of neuronal Cl⁻ homeostasis.

http://www.wikidata.org/entity/Q37152999

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Neurobiology of opioid dependence in creating addiction vulnerability Glia as drivers of abnormal neuronal activity Pain and Poppies: The Good, the Bad, and the Ugly of Opioid Analgesics Opioid-induced central immune signaling: implications for opioid analgesia Emerging targets in neuroinflammation-driven chronic pain Mu opioid receptors on primary afferent nav1.8 neurons contribute to opiate-induced analgesia: insight from conditional knockout mice Activation of mu opioid receptors sensitizes transient receptor potential vanilloid type 1 (TRPV1) via β-arrestin-2-mediated cross-talk Opioid receptor-triggered spinal mTORC1 activation contributes to morphine tolerance and hyperalgesia Neuroinflammation-a co-occurring phenomenon linking chronic pain and opioid dependence.Toll-like receptor 4 mutant and null mice retain morphine-induced tolerance, hyperalgesia, and physical dependence.The Netrin-1 receptor DCC is a regulator of maladaptive responses to chronic morphine administration Brain-derived neurotrophic factor involved epigenetic repression of UGT2B7 in colorectal carcinoma: A mechanism to alter morphine glucuronidation in tumor.P2X4 Receptor Function in the Nervous System and Current Breakthroughs in Pharmacology.Biphalin, a Dimeric Enkephalin, Alleviates LPS-Induced Activation in Rat Primary Microglial Cultures in Opioid Receptor-Dependent and Receptor-Independent Manners.Targeting brain-derived neurotrophic factor in the medial thalamus for the treatment of central poststroke pain in a rodent model.β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain.Pain regulation by non-neuronal cells and inflammation.Pathological pain and the neuroimmune interface.β-arrestin-2-biased agonism of delta opioid receptors sensitizes transient receptor potential vanilloid type 1 (TRPV1) in primary sensory neurons.Erythropoietin attenuates loss of potassium chloride co-transporters following prenatal brain injury.Epigenetic regulation of spinal cord gene expression controls opioid-induced hyperalgesia.NADPH-oxidase 2 activation promotes opioid-induced antinociceptive tolerance in mice.Glial TLR4 signaling does not contribute to opioid-induced depression of respiration.Chloride extrusion enhancers as novel therapeutics for neurological diseases Toward a systematic approach to opioid rotation.Prevalence of narcotic bowel syndrome in opioid abusers in iran.A spinal analog of memory reconsolidation enables reversal of hyperalgesia.Evidence for brain glial activation in chronic pain patients.The pain interactome: connecting pain-specific protein interactions.Activation of adult rat CNS endothelial cells by opioid-induced toll-like receptor 4 (TLR4) signaling induces proinflammatory, biochemical, morphological, and behavioral sequelae Cation-chloride cotransporters in neuronal development, plasticity and disease Reactive astrogliosis causes the development of spontaneous seizures.Role of the outer pore domain in transient receptor potential vanilloid 1 dynamic permeability to large cations Engagement of the GABA to KCC2 signaling pathway contributes to the analgesic effects of A3AR agonists in neuropathic pain Microglia disrupt mesolimbic reward circuitry in chronic pain.Therapeutic restoration of spinal inhibition via druggable enhancement of potassium-chloride cotransporter KCC2-mediated chloride extrusion in peripheral neuropathic pain Intrathecal rapamycin attenuates morphine-induced analgesic tolerance and hyperalgesia in rats with neuropathic pain.High salt intake increases blood pressure via BDNF-mediated downregulation of KCC2 and impaired baroreflex inhibition of vasopressin neurons Assessment of morphine-induced hyperalgesia and analgesic tolerance in mice using thermal and mechanical nociceptive modalities.Neuroimmune Regulation of GABAergic Neurons Within the Ventral Tegmental Area During Withdrawal from Chronic Morphine.

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Morphine hyperalgesia gated through microglia-mediated disruption of neuronal Cl⁻ homeostasis.

http://www.wikidata.org/entity/Q37152999

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 January 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

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Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

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type

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Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

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Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

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prefLabel

Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

@en

Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

@nl

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Nature Neuroscience

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Morphine hyperalgesia gated th ...... n of neuronal Cl⁻ homeostasis.

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Antoine G Godin

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Catherine M Cahill

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Karen Vandal

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Louis-Etienne Lorenzo

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Michael W Salter

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Nicolas Doyon

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Simon Beggs

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Sophie Laffray

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Theresa-Alexandra M Mattioli

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Tuan Trang

http://www.w3.org/2001/XMLSchema#string

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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Pain mechanisms: labeled lines versus convergence in central processing

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Mu-opioid receptor desensitization by beta-arrestin-2 determines morphine tolerance but not dependence

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183-192

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scholarly article

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10.1038/NN.3295

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2

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16

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Jean-Martin Beaulieu

Yves De Koninck

Annie Castonguay

Francesco Ferrini

Thomas Del'Guidice

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2013-01-06T00:00:00Z

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234070377

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23292683

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