Misoprostol impairs female reproductive tract innate immunity against Clostridium sordellii.
about
Lethal toxin is a critical determinant of rapid mortality in rodent models of Clostridium sordellii endometritis.Comparative analysis of the extracellular proteomes of two Clostridium sordellii strains exhibiting contrasting virulenceCyclooxygenase-2 enhances antimicrobial peptide expression and killing of Staphylococcus aureus.TcsL is an essential virulence factor in Clostridium sordellii ATCC 9714Progesterone analogs influence germination of Clostridium sordellii and Clostridium difficile spores in vitro.MicroRNA 21 is a homeostatic regulator of macrophage polarization and prevents prostaglandin E2-mediated M2 generation.Clostridium sordellii genome analysis reveals plasmid localized toxin genes encoded within pathogenicity lociPostpartum group a Streptococcus sepsis and maternal immunology.Unusual complication of vaginal delivery: is misoprostal the cause?Eicosanoids and respiratory viral infection: coordinators of inflammation and potential therapeutic targets.Neither vaginal nor buccal administration of 800 μg misoprostol alters mucosal and systemic immune activation or the cervicovaginal microbiome: a pilot study.Prostaglandin E2 and the suppression of phagocyte innate immune responses in different organsLeukotriene B4 enhances innate immune defense against the puerperal sepsis agent Streptococcus pyogenes.A novel murine model of Clostridium sordellii myonecrosis: Insights into the pathogenesis of diseaseAntimicrobial peptide response to group B Streptococcus in human extraplacental membranes in culture.The Sialidase NanS Enhances Non-TcsL Mediated Cytotoxicity of Clostridium sordelliiIntrauterine group A streptococcal infections are exacerbated by prostaglandin E2.Identification and characterization of Clostridium sordellii toxin gene regulator.Establishment of hydrochloric acid/lipopolysaccharide-induced pelvic inflammatory disease model.EP4 and EP2 receptor activation of protein kinase A by prostaglandin E2 impairs macrophage phagocytosis of Clostridium sordellii.Inflammatory Cyclooxygenase Activity and PGE2 Signaling in Models of Alzheimer's DiseaseEvaluation of intravaginal misoprostol for medical management of pregnancies less than 20 weeks of gestation with absent cardiac activity.Clostridium sordellii outer spore proteins maintain spore structural integrity and promote bacterial clearance from the gastrointestinal tract.
P2860
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P2860
Misoprostol impairs female reproductive tract innate immunity against Clostridium sordellii.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on June 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Misoprostol impairs female rep ...... against Clostridium sordellii.
@en
Misoprostol impairs female rep ...... against Clostridium sordellii.
@nl
type
label
Misoprostol impairs female rep ...... against Clostridium sordellii.
@en
Misoprostol impairs female rep ...... against Clostridium sordellii.
@nl
prefLabel
Misoprostol impairs female rep ...... against Clostridium sordellii.
@en
Misoprostol impairs female rep ...... against Clostridium sordellii.
@nl
P2093
P2860
P1476
Misoprostol impairs female rep ...... against Clostridium sordellii.
@en
P2093
Camila M Peres
Carlos H Serezani
Casey Lewis
Gwo-Hsiao Chen
Jooho Chung
Nicolas Flamand
Nicole Coleman
Thomas G Brock
P2860
P304
P356
10.4049/JIMMUNOL.180.12.8222
P407
P577
2008-06-01T00:00:00Z