Upregulation of MHC class I in transgenic mice results in reduced force-generating capacity in slow-twitch muscle. - Wikidata - wikimedia - TriplyDB

Upregulation of MHC class I in transgenic mice results in reduced force-generating capacity in slow-twitch muscle.

Upregulation of MHC class I in transgenic mice results in reduced force-generating capacity in slow-twitch muscle.

http://www.wikidata.org/entity/Q37154934

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Role of non-immune mechanisms of muscle damage in idiopathic inflammatory myopathies Daily supplementation of D-ribose shows no therapeutic benefits in the MHC-I transgenic mouse model of inflammatory myositis Juvenile dermatomyositis: new developments in pathogenesis, assessment and treatment.The molecular basis of skeletal muscle weakness in a mouse model of inflammatory myopathy Developments in the scientific and clinical understanding of inflammatory myopathies.Can muscle regeneration fail in chronic inflammation: a weakness in inflammatory myopathies?Pathogenesis, classification and treatment of inflammatory myopathies.Innate immune-response mechanisms in dermatomyositis: an update on pathogenesis, diagnosis and treatment.In the idiopathic inflammatory myopathies (IIM), do reactive oxygen species (ROS) contribute to muscle weakness?Isometric and eccentric force generation assessment of skeletal muscles isolated from murine models of muscular dystrophies.HMGB1 expression and muscle regeneration in idiopathic inflammatory myopathies and degenerative joint diseases.Increased levels of HSPA5 in the serum of patients with inflammatory myopathies--preliminary findings.

P2860

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P2860

Upregulation of MHC class I in transgenic mice results in reduced force-generating capacity in slow-twitch muscle.

http://www.wikidata.org/entity/Q37154934

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Upregulation of MHC class I in ...... apacity in slow-twitch muscle.

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Muscle and Nerve

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Upregulation of MHC class I in ...... capacity in slow-twitch muscle

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P2093

Abram Katz

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Cecilia Grundtman

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Charles Li

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Shi-Jin Zhang

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Stina Salomonsson

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P2860

Muscle inflammation and MHC class I up-regulation in muscular dystrophy with lack of dysferlin: an immunopathological study

RAGE is the major receptor for the proinflammatory activity of HMGB1 in rodent macrophages

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

Characteristics of the dephosphorylated form of phosphorylase purified from rat liver and measurement of its activity in crude liver preparations.

Skeletal muscle fibers express major histocompatibility complex class II antigens independently of inflammatory infiltrates in inflammatory myopathies.

Conditional up-regulation of MHC class I in skeletal muscle leads to self-sustaining autoimmune myositis and myositis-specific autoantibodies

Expression of immunoreactive major histocompatibility complex products in human skeletal muscles.

Free radical-mediated skeletal muscle dysfunction in inflammatory conditions.

Oxidative stress, chronic disease, and muscle wasting.

Impaired calcium release during fatigue.

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674-682

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scholarly article

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10.1002/MUS.21129

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5

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39

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Kanneboyina Nagaraju

Ingrid E Lundberg

Hakan Westerblad

Johanna T Lanner

Lucy R Wedderburn

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2009-05-01T00:00:00Z

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24027814

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19229963

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2667561

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