Matrix metalloproteinase-9 contributes to brain extravasation and edema in fulminant hepatic failure mice. - Wikidata - wikimedia - TriplyDB

Matrix metalloproteinase-9 contributes to brain extravasation and edema in fulminant hepatic failure mice.

Matrix metalloproteinase-9 contributes to brain extravasation and edema in fulminant hepatic failure mice.

http://www.wikidata.org/entity/Q37155284

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Alterations of blood brain barrier function in hyperammonemia: an overview Neuroinflammation in hepatic encephalopathy: mechanistic aspects Brain magnetic resonance spectroscopy in episodic hepatic encephalopathy.Neuronal CCL2 is upregulated during hepatic encephalopathy and contributes to microglia activation and neurological decline.Occludin is regulated by epidermal growth factor receptor activation in brain endothelial cells and brains of mice with acute liver failure.Ammonia increases paracellular permeability of rat brain endothelial cells by a mechanism encompassing oxidative/nitrosative stress and activation of matrix metalloproteinases.Matrix metalloproteinases in liver injury, repair and fibrosis.Matrix metalloproteinase-9 contributes to parenchymal hemorrhage and necrosis in the remnant liver after extended hepatectomy in mice Bile Acid Signaling Is Involved in the Neurological Decline in a Murine Model of Acute Liver Failure.Brain edema in acute liver failure: role of neurosteroids.Real-time analysis of microglial activation and motility in hepatic and hyperammonemic encephalopathy.Brain edema in acute liver failure: can it be prevented? Can it be treated?TIMP-1 attenuates blood-brain barrier permeability in mice with acute liver failure.Mechanisms of brain edema in acute liver failure and impact of novel therapeutic interventions.Altered expression of zonula occludens-2 precedes increased blood-brain barrier permeability in a murine model of fulminant hepatic failure TGFβ1 exacerbates blood-brain barrier permeability in a mouse model of hepatic encephalopathy via upregulation of MMP9 and downregulation of claudin-5 Glycosylation patterns and PHA-E-associated glycoprotein profiling associated with early hepatic encephalopathy in Chinese hepatocellular carcinoma patients.Pathophysiology of cerebral oedema in acute liver failure.Oxidative stress and extracellular matrices after hepatectomy and liver transplantation in rats Blood-brain barrier in acute liver failure Endothelial-astrocytic interactions in acute liver failure.Brain edema in acute liver failure: mechanisms and concepts.Fulminant liver failure model with hepatic encephalopathy in the mouse.Free Base Lysine Increases Survival and Reduces Metastasis in Prostate Cancer Model.Hypothermia predicts hepatic failure after extensive hepatectomy in mice.Ammonia-induced brain swelling and neurotoxicity in an organotypic slice model.Alterations of blood-brain barrier and associated factors in acute liver failure.Tumor necrosis factor-alpha-induced reduction of glomerular filtration rate in rats with fulminant hepatic failure.The association between FABP7 serum levels with survival and neurological complications in acetaminophen-induced acute liver failure: a nested case-control study.Subtle BBB alterations in brain edema associated with acute liver failure.Disruptions of occludin and claudin-5 in brain endothelial cells in vitro and in brains of mice with acute liver failure.Lipopolysaccharide precipitates hepatic encephalopathy and increases blood-brain barrier permeability in mice with acute liver failure.Ammonia promotes endothelial cell survival via the heme oxygenase-1-mediated release of carbon monoxide.Increased Th1 immune response in SERPINB3 transgenic mice during acute liver failure.Extracellular matrix metalloproteinase inducer is associated with severity of brain oedema following experimental subarachnoid haemorrhage in rats.Regional reduction in gray and white matter volume in brains of cirrhotic patients: voxel-based analysis of MRI.Gene expression profiling of brain cortex microvessels may support brain vasodilation in acute liver failure rat models.Lysophosphatidic acid receptor, LPA6, regulates endothelial blood-brain barrier function: Implication of hepatic encephalopathy.FXR-Mediated Cortical Cholesterol Accumulation Contributes to the Pathogenesis of Type A Hepatic Encephalopathy.Pathogenesis of hepatic encephalopathy

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Matrix metalloproteinase-9 contributes to brain extravasation and edema in fulminant hepatic failure mice.

http://www.wikidata.org/entity/Q37155284

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2005 nî lūn-bûn

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2005年学术文章

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2005年學術文章

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Matrix metalloproteinase-9 con ...... ulminant hepatic failure mice.

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Matrix metalloproteinase-9 con ...... ulminant hepatic failure mice.

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Matrix metalloproteinase-9 con ...... ulminant hepatic failure mice.

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Matrix metalloproteinase-9 con ...... ulminant hepatic failure mice.

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J.JHEP.2005.09.019

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Journal of Hepatology

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Matrix metalloproteinase-9 con ...... ulminant hepatic failure mice.

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Christopher B Eckman

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Daniel Sevlever

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Elliott Richelson

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Jeffery Steers

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Justin H Nguyen

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Michael McKinney

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Monica Castanedes-Casey

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Naoki Shimojima

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Petrina Genco

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Satoshi Yamamoto

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P2860

How matrix metalloproteinases regulate cell behavior

Matrix metalloproteinase 2 gene knockout has no effect on acute brain injury after focal ischemia.

Cortical spreading depression activates and upregulates MMP-9.

Brain edema in liver failure: basic physiologic principles and management.

The management of fulminant hepatic failure.

Molecular physiology and pathophysiology of tight junctions in the blood-brain barrier.

Acute liver failure in the United States.

Etiology and management of fulminant hepatic failure.

Reversal of experimental autoimmune encephalomyelitis with a hydroxamate inhibitor of matrix metalloproteases

The normal and pathological physiology of brain water.

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