Intralipid, a clinically safe compound, protects the heart against ischemia-reperfusion injury more efficiently than cyclosporine-A. - Wikidata - wikimedia - TriplyDB

Intralipid, a clinically safe compound, protects the heart against ischemia-reperfusion injury more efficiently than cyclosporine-A.

Intralipid, a clinically safe compound, protects the heart against ischemia-reperfusion injury more efficiently than cyclosporine-A.

http://www.wikidata.org/entity/Q37160450

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The mechanism of Intralipid®-mediated cardioprotection complex IV inhibition by the active metabolite, palmitoylcarnitine, generates reactive oxygen species and activates reperfusion injury salvage kinases 9th Hatter Biannual Meeting: position document on ischaemia/reperfusion injury, conditioning and the ten commandments of cardioprotection Loss of Intralipid®- but not sevoflurane-mediated cardioprotection in early type-2 diabetic hearts of fructose-fed rats: importance of ROS signaling The number of X chromosomes influences protection from cardiac ischaemia/reperfusion injury in mice: one X is better than two.Resuscitation with lipid emulsion: dose-dependent recovery from cardiac pharmacotoxicity requires a cardiotonic effect Closing the pore on reperfusion injury: myocardial protection with cyclosporine Signaling epicenters: the role of caveolae and caveolins in volatile anesthetic induced cardiac protection.Lipid emulsion rapidly restores contractility in stunned mouse cardiomyocytes: a comparison with therapeutic hypothermia.Acute administration of n-3 rich triglyceride emulsions provides cardioprotection in murine models after ischemia-reperfusion G Protein-Coupled Estrogen Receptor 1 Mediates Acute Estrogen-Induced Cardioprotection via MEK/ERK/GSK-3β Pathway after Ischemia/Reperfusion.Effects of Cyclosporine on Reperfusion Injury in Patients: A Meta-Analysis of Randomized Controlled Trials.Enhancing Cardiac Triacylglycerol Metabolism Improves Recovery From Ischemic Stress.Morphine-Induced Preconditioning: Involvement of Protein Kinase A and Mitochondrial Permeability Transition Pore Meeting report from the 2nd International Symposium on New Frontiers in Cardiovascular Research. Protecting the cardiovascular system from ischemia: between bench and bedside Insulin Signaling in Bupivacaine-induced Cardiac Toxicity: Sensitization during Recovery and Potentiation by Lipid Emulsion Adding Emulsified Isoflurane to Cardioplegia Solution Produces Cardiac Protection in a Dog Cardiopulmonary Bypass Model Tilianin Post-Conditioning Attenuates Myocardial Ischemia/Reperfusion Injury via Mitochondrial Protection and Inhibition of Apoptosis.Intralipid: the new magic bullet in cardioprotection?Intralipid protects the heart in late pregnancy against ischemia/reperfusion injury via Caveolin2/STAT3/GSK-3β pathway.Lipid emulsion enhances cardiac performance after ischemia-reperfusion in isolated hearts from summer-active arctic ground squirrels.

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Intralipid, a clinically safe compound, protects the heart against ischemia-reperfusion injury more efficiently than cyclosporine-A.

http://www.wikidata.org/entity/Q37160450

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articolo scientifico

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bilimsel makale

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scientific article published on October 2012

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vedecký článok

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vetenskaplig artikel

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vědecký článek

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name

Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Intralipid, a clinically safe ...... ficiently than cyclosporine-A.

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Andrea Iorga

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Hua Cai

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Ji-Youn Youn

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Jingyuan Li

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Mansoureh Eghbali

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Rod Partow-Navid

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Salil Sharma

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Siamak Rahman

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Soban Umar

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P2860

Netrin-1 prevents ischemia/reperfusion-induced myocardial infarction via a DCC/ERK1/2/eNOS s1177/NO/DCC feed-forward mechanism

The mitochondrial permeability transition pore and its role in cell death

Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes.

A novel estrogen receptor GPER inhibits mitochondria permeability transition pore opening and protects the heart against ischemia-reperfusion injury.

Cyclosporine A at reperfusion reduces infarct size in pigs

Endothelial dihydrofolate reductase: critical for nitric oxide bioavailability and role in angiotensin II uncoupling of endothelial nitric oxide synthase

Role of mitochondrial inner membrane permeabilization in necrotic cell death, apoptosis, and autophagy.

Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection.

Phosphorylation of GSK-3β mediates intralipid-induced cardioprotection against ischemia/reperfusion injury.

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836-846

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reperfusion injury

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