Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation. - Wikidata - wikimedia - TriplyDB

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

http://www.wikidata.org/entity/Q37163219

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Effects of PTH on osteocyte function Regulation of postnatal bone homeostasis by TGFβ Recent progress in osteocyte research Osteocytes: master orchestrators of bone Parathyroid Hormone Induces Bone Cell Motility and Loss of Mature Osteocyte Phenotype through L-Calcium Channel Dependent and Independent Mechanisms Novel approaches for two and three dimensional multiplexed imaging of osteocytes.FGF-21 and skeletal remodeling during and after lactation in C57BL/6J mice.Structural and mechanical repair of diffuse damage in cortical bone in vivo Disrupted bone remodeling leads to cochlear overgrowth and hearing loss in a mouse model of fibrous dysplasia.Alterations of mass density and 3D osteocyte lacunar properties in bisphosphonate-related osteonecrotic human jaw bone, a synchrotron µCT study.Total water, phosphorus relaxation and inter-atomic organic to inorganic interface are new determinants of trabecular bone integrity Excessive dietary intake of vitamin A reduces skull bone thickness in mice.Administration frequency as well as dosage of PTH are associated with development of cortical porosity in ovariectomized rats.Vertebral bone microarchitecture and osteocyte characteristics of three toothed whale species with varying diving behaviour.Intraskeletal variation in human cortical osteocyte lacunar density: Implications for bone quality assessment.Biological regulation of bone quality.Increased tartrate-resistant Acid phosphatase expression in osteoblasts and osteocytes in experimental osteoporosis in rats.Redefining Human Vitamin D Sufficiency: Back to the Basics Architecture of the osteocyte network correlates with bone material quality.Upregulation of calcitriol during pregnancy and skeletal recovery after lactation do not require parathyroid hormone.Microgravity induces pelvic bone loss through osteoclastic activity, osteocytic osteolysis, and osteoblastic cell cycle inhibition by CDKN1a/p21.Talbot-defocus multiscan tomography using the synchrotron X-ray microscope to study the lacuno-canalicular network in mouse bone Unexpected long-term effects of calcium supplementation in pregnancy on maternal bone outcomes in women with a low calcium intake: a follow-up study.Tartrate-resistant acid phosphatase (TRAP) co-localizes with receptor activator of NF-KB ligand (RANKL) and osteoprotegerin (OPG) in lysosomal-associated membrane protein 1 (LAMP1)-positive vesicles in rat osteoblasts and osteocytes.Periostin deficiency increases bone damage and impairs injury response to fatigue loading in adult mice Evidence for the role of connexin 43-mediated intercellular communication in the process of intracortical bone resorption via osteocytic osteolysis Ovariectomy enhances mechanical load-induced solute transport around osteocytes in rat cancellous bone.Transcriptional Profiling of Laser Capture Microdissected Subpopulations of the Osteoblast Lineage Provides Insight Into the Early Response to Sclerostin Antibody in Rats.Update on type 2 diabetes-related osteoporosis.Plasma fluctuation in estradiol-17β and bone resorption markers around parturition in dairy cows.Impact of Non-Invasively Induced Motor Deficits on Tibial Cortical Properties in Mutant Lurcher Mice Beta-Catenin Haplo Insufficient Male Mice Do Not Lose Bone in Response to Hindlimb Unloading.Alterations in the osteocyte lacunar-canalicular microenvironment due to estrogen deficiency.Matrix metalloproteinase-13 is required for osteocytic perilacunar remodeling and maintains bone fracture resistance.Bone marrow oxytocin mediates the anabolic action of estrogen on the skeleton.Osteocytes and Skeletal Pathophysiology Parathyroid hormone 1 receptor is essential to induce FGF23 production and maintain systemic mineral ion homeostasis.Current concepts in perinatal mineral metabolism PTHrP regulates the modeling of cortical bone surfaces at fibrous insertion sites during growth.Osteocytes remove and replace perilacunar mineral during reproductive cycles.

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P2860

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

http://www.wikidata.org/entity/Q37163219

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on May 2012

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@en

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@nl

type

label

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@en

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@nl

prefLabel

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@en

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation.

@nl

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P1433

Journal of Bone and Mineral Research

P1476

Demonstration of osteocytic perilacunar/canalicular remodeling in mice during lactation

@en

P2093

Hai Qing

http://www.w3.org/2001/XMLSchema#string

John Wysolmerski

http://www.w3.org/2001/XMLSchema#string

Laleh Ardeshirpour

http://www.w3.org/2001/XMLSchema#string

Lynda F Bonewald

http://www.w3.org/2001/XMLSchema#string

Paola Divieti Pajevic

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Shigeaki Kato

http://www.w3.org/2001/XMLSchema#string

Vladimir Dusevich

http://www.w3.org/2001/XMLSchema#string

P2860

The amazing osteocyte

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Generalized lacZ expression with the ROSA26 Cre reporter strain

Sclerostin is a delayed secreted product of osteocytes that inhibits bone formation

Osteocyte ultrastructure in renal osteodystrophy.

Gap junctions and biophysical regulation of bone cell differentiation.

Increased PTHrP and decreased estrogens alter bone turnover but do not reproduce the full effects of lactation on the skeleton.

Estrogen prevents bone loss via estrogen receptor alpha and induction of Fas ligand in osteoclasts.

Glucocorticoid-treated mice have localized changes in trabecular bone material properties and osteocyte lacunar size that are not observed in placebo-treated or estrogen-deficient mice.

Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.

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1018-1029

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scholarly article

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10.1002/JBMR.1567

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5

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27

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Katharina Jähn

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2012-05-01T00:00:00Z

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221808526

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22308018

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3770147

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