High glucose suppresses epidermal growth factor receptor/phosphatidylinositol 3-kinase/Akt signaling pathway and attenuates corneal epithelial wound healing.
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Exogenous endophthalmitis in diabetic patients: a systemic reviewProgress in corneal wound healingAdenovirus-driven overexpression of proteinases in organ-cultured normal human corneas leads to diabetic-like changesTopical application of naltrexone facilitates reepithelialization of the cornea in diabetic rabbitsNormalization of wound healing and diabetic markers in organ cultured human diabetic corneas by adenoviral delivery of c-Met geneDiabetic keratopathy and treatment by modulation of the opioid growth factor (OGF)-OGF receptor (OGFr) axis with naltrexone: a reviewLL-37 via EGFR transactivation to promote high glucose-attenuated epithelial wound healing in organ-cultured corneas.High glucose promotes pancreatic cancer cell proliferation via the induction of EGF expression and transactivation of EGFRFIH-1 disrupts an LRRK1/EGFR complex to positively regulate keratinocyte migration.Normalization of wound healing and stem cell marker patterns in organ-cultured human diabetic corneas by gene therapy of limbal cells.Targeting miR-146a to treat delayed wound healing in human diabetic organ-cultured corneasAdvanced glycation end products induce human corneal epithelial cells apoptosis through generation of reactive oxygen species and activation of JNK and p38 MAPK pathways.Knockdown of heme oxygenase-2 impairs corneal epithelial cell wound healingLow levels of hydrogen peroxide stimulate corneal epithelial cell adhesion, migration, and wound healing.Impaired epithelial wound healing and EGFR signaling pathways in the corneas of diabetic rats.Differentially expressed wound healing-related microRNAs in the human diabetic cornea.Comprehensive Review of the Effects of Diabetes on Ocular Health.Alterations of epithelial stem cell marker patterns in human diabetic corneas and effects of c-met gene therapyCorneal complications in streptozocin-induced type I diabetic rats.Dendritic cell-epithelium interplay is a determinant factor for corneal epithelial wound repair.SIRT1 is a regulator in high glucose-induced inflammatory response in RAW264.7 cells.Human growth hormone promotes corneal epithelial cell migration in vitroEphA2/Ephrin-A1 signaling complexes restrict corneal epithelial cell migration.MicroRNA-31 targets FIH-1 to positively regulate corneal epithelial glycogen metabolism.Upregulation of TLR4 via PKC activation contributes to impaired wound healing in high-glucose-treated kidney proximal tubular cellsReduced innervation and delayed re-innervation after epithelial wounding in type 2 diabetic Goto-Kakizaki ratsPersistence of reduced expression of putative stem cell markers and slow wound healing in cultured diabetic limbal epithelial cells.Dendritic cell dysfunction and diabetic sensory neuropathy in the corneaFOXO1 promotes wound healing through the up-regulation of TGF-β1 and prevention of oxidative stress.Enhanced wound healing, kinase and stem cell marker expression in diabetic organ-cultured human corneas upon MMP-10 and cathepsin F gene silencing.Genome-wide transcriptional analysis of differentially expressed genes in diabetic, healing corneal epithelial cells: hyperglycemia-suppressed TGFβ3 expression contributes to the delay of epithelial wound healing in diabetic corneas.A systematic review on the impact of diabetes mellitus on the ocular surface.Epidermal growth factor receptor in corneal damage: update and new insights from recent reports.Impact of diabetes on alpha-crystallins and other heat shock proteins in the eye.Unravelling the stromal-nerve interactions in the human diabetic cornea.Biochemical and Biophysical Cues in Matrix Design for Chronic and Diabetic Wound Treatment.Diabetic complications in the cornea.Sensory nerve regeneration after epithelium wounding in normal and diabetic cornea.Adenoviral Gene Therapy for Diabetic Keratopathy: Effects on Wound Healing and Stem Cell Marker Expression in Human Organ-cultured Corneas and Limbal Epithelial CellsEfficacy of Sodium Hyaluronate in Murine Diabetic Ocular Surface Diseases.
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High glucose suppresses epidermal growth factor receptor/phosphatidylinositol 3-kinase/Akt signaling pathway and attenuates corneal epithelial wound healing.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 02 February 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
High glucose suppresses epider ...... neal epithelial wound healing.
@en
High glucose suppresses epider ...... neal epithelial wound healing.
@nl
type
label
High glucose suppresses epider ...... neal epithelial wound healing.
@en
High glucose suppresses epider ...... neal epithelial wound healing.
@nl
prefLabel
High glucose suppresses epider ...... neal epithelial wound healing.
@en
High glucose suppresses epider ...... neal epithelial wound healing.
@nl
P2093
P2860
P356
P1433
P1476
High glucose suppresses epider ...... neal epithelial wound healing.
@en
P2093
Alexander V Ljubimov
Fu-Shin X Yu
Ke-Ping Xu
Yanfeng Li
P2860
P304
P356
10.2337/DB08-0997
P407
P577
2009-02-02T00:00:00Z