The fast and slow afterhyperpolarizations are differentially modulated in hippocampal neurons by aging and learning.
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Dissecting mechanisms of brain aging by studying the intrinsic excitability of neuronsIntrinsic Hippocampal Excitability Changes of Opposite Signs and Different Origins in CA1 and CA3 Pyramidal Neurons Underlie Aging-Related Cognitive DeficitsThe impact of hippocampal lesions on trace-eyeblink conditioning and forebrain-cerebellar interactionsIncreased Excitability of Both Principal Neurons and Interneurons during Associative LearningHigh-fat diet impairs spatial memory and hippocampal intrinsic excitability and sex-dependently alters circulating insulin and hippocampal insulin sensitivityChronic cocaine disrupts mesocortical learning mechanismsResonant Cholinergic Dynamics in Cognitive and Motor Decision-Making: Attention, Category Learning, and Choice in Neocortex, Superior Colliculus, and Optic Tectum.Mechanisms underlying basal and learning-related intrinsic excitability in a mouse model of Alzheimer's disease.Evidence for altered hippocampal function in a mouse model of the human 22q11.2 microdeletion.Learning and aging related changes in intrinsic neuronal excitabilityPlasma membrane sphingomyelin hydrolysis increases hippocampal neuron excitability by sphingosine-1-phosphate mediated mechanisms.Age-related increase of sI(AHP) in prefrontal pyramidal cells of monkeys: relationship to cognition.Redox sensitive calcium stores underlie enhanced after hyperpolarization of aged neurons: role for ryanodine receptor mediated calcium signalingDietary cholesterol modulates the excitability of rabbit hippocampal CA1 pyramidal neurons.Modulation of BK channels contributes to activity-dependent increase of excitability through MTORC1 activity in CA1 pyramidal cells of mouse hippocampusOf mice and intrinsic excitability: genetic background affects the size of the postburst afterhyperpolarization in CA1 pyramidal neurons.Age-related changes to layer 3 pyramidal cells in the rhesus monkey visual cortex.Efferent Vestibular Neurons Show Homogenous Discharge Output But Heterogeneous Synaptic Input Profile In Vitro.Physiology and pathology of calcium signaling in the brain.Aging-Related Hyperexcitability in CA3 Pyramidal Neurons Is Mediated by Enhanced A-Type K+ Channel Function and Expression.Characterizing cognitive aging of associative memory in animal models.Genome-wide transcriptomics of aging in the rotifer Brachionus manjavacas, an emerging model system.Physiological and anatomical studies of associative learning: Convergence with learning studies of W.T. Greenough.Altered calcium metabolism in aging CA1 hippocampal pyramidal neuronsLinking redox regulation of NMDAR synaptic function to cognitive decline during aging.Learning to learn - intrinsic plasticity as a metaplasticity mechanism for memory formation.L-type Ca2+ currents at CA1 synapses, but not CA3 or dentate granule neuron synapses, are increased in 3xTgAD mice in an age-dependent manner.CREB overexpression in dorsal CA1 ameliorates long-term memory deficits in aged rats.Surface L-type Ca2+ channel expression levels are increased in aged hippocampus.Taste-related sensations in old age.Nutritional strategies to optimise cognitive function in the aging brain.Dopamine terminals from the ventral tegmental area gate intrinsic inhibition in the prefrontal cortex.Electrical and Network Neuronal Properties Are Preferentially Disrupted in Dorsal, But Not Ventral, Medial Entorhinal Cortex in a Mouse Model of Tauopathy.CA1 pyramidal cells have diverse biophysical properties, affected by development, experience, and agingAfter-hyperpolarization currents and acetylcholine control sigmoid transfer functions in a spiking cortical model.Cortical nNOS/NK1 Receptor Neurons are Regulated by Cholinergic Projections From the Basal Forebrain.Calcium homeostasis in aging neurons.Dynamic DNA methylation regulates neuronal intrinsic membrane excitability.Similar nicotinic excitability responses across the developing hippocampal formation are regulated by small conductance calcium-activated potassium channels.Learning-dependent plasticity of hippocampal CA1 pyramidal neuron postburst afterhyperpolarizations and increased excitability after inhibitory avoidance learning depend upon basolateral amygdala inputs.
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The fast and slow afterhyperpolarizations are differentially modulated in hippocampal neurons by aging and learning.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
The fast and slow afterhyperpo ...... neurons by aging and learning.
@en
The fast and slow afterhyperpo ...... neurons by aging and learning.
@nl
type
label
The fast and slow afterhyperpo ...... neurons by aging and learning.
@en
The fast and slow afterhyperpo ...... neurons by aging and learning.
@nl
prefLabel
The fast and slow afterhyperpo ...... neurons by aging and learning.
@en
The fast and slow afterhyperpo ...... neurons by aging and learning.
@nl
P2093
P2860
P1476
The fast and slow afterhyperpo ...... neurons by aging and learning.
@en
P2093
Elizabeth A Matthews
John F Disterhoft
John M Linardakis
P2860
P304
P356
10.1523/JNEUROSCI.0384-09.2009
P407
P577
2009-04-01T00:00:00Z