Effect of Urinary Kallidinogenase on Transforming Growth Factor-β1 and High-Sensitivity C-Reactive Protein Expression in Rat Focal Cerebral Ischemic Injury

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Therapeutic Values of Human Urinary Kallidinogenase on Cerebrovascular Diseases.

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Q55177578-C9D87775-7FD7-44F3-8735-68270C2A5CD3

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Effect of Urinary Kallidinogenase on Transforming Growth Factor-β1 and High-Sensitivity C-Reactive Protein Expression in Rat Focal Cerebral Ischemic Injury

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article científic

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bilimsel makale

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scientific article published on 13 August 2016

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vědecký článek

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Effect of Urinary Kallidinogen ...... Focal Cerebral Ischemic Injury

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Effect of Urinary Kallidinogen ...... ocal Cerebral Ischemic Injury.

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Effect of Urinary Kallidinogen ...... Focal Cerebral Ischemic Injury

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Effect of Urinary Kallidinogen ...... ocal Cerebral Ischemic Injury.

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Effect of Urinary Kallidinogen ...... Focal Cerebral Ischemic Injury

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Effect of Urinary Kallidinogen ...... ocal Cerebral Ischemic Injury.

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Effect of Urinary Kallidinogen ...... Focal Cerebral Ischemic Injury

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Hai-Qing Yan

http://www.w3.org/2001/XMLSchema#string

Hai-Xia Lv

http://www.w3.org/2001/XMLSchema#string

Ping Zhang

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Ting-Fang Dong

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Tong Li

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Xiao-Lu Niu

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Yong-Kun Gui

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International union of pharmacology. XLV. Classification of the kinin receptor family: from molecular mechanisms to pathophysiological consequences

Attenuated inflammatory response in aged mice brains following stroke

Reduction of inflammatory response in the mouse brain with adenoviral-mediated transforming growth factor-ss1 expression.

Intranasal delivery of transforming growth factor-beta1 in mice after stroke reduces infarct volume and increases neurogenesis in the subventricular zone

Human urinary kallidinogenase suppresses cerebral inflammation in experimental stroke and downregulates nuclear factor-kappaB.

Time course, distribution and cell types of induction of transforming growth factor betas following middle cerebral artery occlusion in the rat brain

Microglial activation state exerts a biphasic influence on brain endothelial cell proliferation by regulating the balance of TNF and TGF-β1.

Transforming growth factor-beta: a neuroprotective factor in cerebral ischemia.

Angiogenesis therapy with human tissue kallikrein for the treatment of ischemic diseases.

Potent anti-inflammatory and neuroprotective effects of TGF-beta1 are mediated through the inhibition of ERK and p47phox-Ser345 phosphorylation and translocation in microglia.

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2852-2858

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scholarly article

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10.12659/MSM.892724

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2016-08-13T00:00:00Z

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27521289

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4988362

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Effect of Urinary Kallidinogenase on Transforming Growth Factor-β1 and High-Sensitivity C-Reactive Protein Expression in Rat Focal Cerebral Ischemic Injury

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P2860

P2860

Effect of Urinary Kallidinogenase on Transforming Growth Factor-β1 and High-Sensitivity C-Reactive Protein Expression in Rat Focal Cerebral Ischemic Injury

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P1476

P2093

P2860

P304

P31

P356

P478

P577

P698

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P356

P1433

P1476

P2093

P2860

P304

P31

P356

P478

P577

P698

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