IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C. - Wikidata - wikimedia - TriplyDB

IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C.

IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C.

http://www.wikidata.org/entity/Q37184166

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IRF3 deficiency impacts granzyme B expression and maintenance of memory T cell function in response to viral infection.Poly(I:C) increases the expression of mPGES-1 and COX-2 in rat primary microglia Interferon β (IFN-β) Production during the Double-stranded RNA (dsRNA) Response in Hepatocytes Involves Coordinated and Feedforward Signaling through Toll-like Receptor 3 (TLR3), RNA-dependent Protein Kinase (PKR), Inducible Nitric Oxide Synthase (i Significant role for IRF3 in both T cell and APC effector functions during T cell responses.Activation of Toll-like receptor 3 increases mouse aortic vascular smooth muscle cell contractility through ERK1/2 pathway.Double-stranded RNA promotes CTL-independent tumor cytolysis mediated by CD11b+Ly6G+ intratumor myeloid cells through the TICAM-1 signaling pathway.Induction of a robust immune response against avian influenza virus following transdermal inoculation with H5-DNA vaccine formulated in modified dendrimer-based delivery system in mouse model.

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IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C.

http://www.wikidata.org/entity/Q37184166

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 23 July 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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J.FEBSLET.2013.07.025

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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.

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Thomas M Petro

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Tyler C Moore

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Toll-like receptor 3-mediated activation of NF-kappaB and IRF3 diverges at Toll-IL-1 receptor domain-containing adapter inducing IFN-beta

Scansite 2.0: Proteome-wide prediction of cell signaling interactions using short sequence motifs

Fast, scalable generation of high-quality protein multiple sequence alignments using Clustal Omega

IKKepsilon and TBK1 are essential components of the IRF3 signaling pathway

Triggering the interferon antiviral response through an IKK-related pathway

The nitric oxide pathway provides innate antiviral protection in conjunction with the type I interferon pathway in fibroblasts

Toll-like receptors and innate immunity

Distinct and essential roles of transcription factors IRF-3 and IRF-7 in response to viruses for IFN-alpha/beta gene induction

Regulation of influenza A virus induced CXCL-10 gene expression requires PI3K/Akt pathway and IRF3 transcription factor.

Inducible nitric oxide contributes to viral pathogenesis following highly pathogenic influenza virus infection in mice.

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10.1016/J.FEBSLET.2013.07.025

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