IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C.
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IRF3 deficiency impacts granzyme B expression and maintenance of memory T cell function in response to viral infection.Poly(I:C) increases the expression of mPGES-1 and COX-2 in rat primary microgliaInterferon β (IFN-β) Production during the Double-stranded RNA (dsRNA) Response in Hepatocytes Involves Coordinated and Feedforward Signaling through Toll-like Receptor 3 (TLR3), RNA-dependent Protein Kinase (PKR), Inducible Nitric Oxide Synthase (iSignificant role for IRF3 in both T cell and APC effector functions during T cell responses.Activation of Toll-like receptor 3 increases mouse aortic vascular smooth muscle cell contractility through ERK1/2 pathway.Double-stranded RNA promotes CTL-independent tumor cytolysis mediated by CD11b+Ly6G+ intratumor myeloid cells through the TICAM-1 signaling pathway.Induction of a robust immune response against avian influenza virus following transdermal inoculation with H5-DNA vaccine formulated in modified dendrimer-based delivery system in mouse model.
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IRF3 and ERK MAP-kinases control nitric oxide production from macrophages in response to poly-I:C.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 July 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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type
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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IRF3 and ERK MAP-kinases contr ...... hages in response to poly-I:C.
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Thomas M Petro
Tyler C Moore
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10.1016/J.FEBSLET.2013.07.025
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2013-07-23T00:00:00Z