Sildenafil attenuates pulmonary inflammation and fibrin deposition, mortality and right ventricular hypertrophy in neonatal hyperoxic lung injury. - Wikidata - wikimedia - TriplyDB

Sildenafil attenuates pulmonary inflammation and fibrin deposition, mortality and right ventricular hypertrophy in neonatal hyperoxic lung injury.

Sildenafil attenuates pulmonary inflammation and fibrin deposition, mortality and right ventricular hypertrophy in neonatal hyperoxic lung injury.

http://www.wikidata.org/entity/Q37190561

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An update on pharmacologic approaches to bronchopulmonary dysplasia Computational drug repositioning for peripheral arterial disease: prediction of anti-inflammatory and pro-angiogenic therapeutics.Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.The Future of Bronchopulmonary Dysplasia: Emerging Pathophysiological Concepts and Potential New Avenues of Treatment.Hydrocortisone normalizes phosphodiesterase-5 activity in pulmonary artery smooth muscle cells from lambs with persistent pulmonary hypertension of the newborn.Novel mechanisms of sildenafil in pulmonary hypertension involving cytokines/chemokines, MAP kinases and Akt.Sildenafil promotes smooth muscle preservation and ameliorates fibrosis through modulation of extracellular matrix and tissue growth factor gene expression after bilateral cavernosal nerve resection in the rat The effect of PPE-induced emphysema and chronic LPS-induced pulmonary inflammation on atherosclerosis development in APOE*3-LEIDEN mice.Drug therapy for the prevention and treatment of bronchopulmonary dysplasia Pharmacokinetics and tolerability of oral sildenafil in adults with cystic fibrosis lung disease Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasia Metformin attenuates hyperoxia-induced lung injury in neonatal rats by reducing the inflammatory response Phosphodiesterase 4 inhibition attenuates persistent heart and lung injury by neonatal hyperoxia in rats.Sildenafil attenuates vaso-obliteration and neovascularization in a mouse model of retinopathy of prematurity.Endothelial actions of atrial and B-type natriuretic peptides Hyperoxia-Induced Proliferative Retinopathy: Early Interruption of Retinal Vascular Development with Severe and Irreversible Neurovascular Disruption.Deficiency or inhibition of lysophosphatidic acid receptor 1 protects against hyperoxia-induced lung injury in neonatal rats.Ambrisentan reduces pulmonary arterial hypertension but does not stimulate alveolar and vascular development in neonatal rats with hyperoxic lung injury Agonists of MAS oncogene and angiotensin II type 2 receptors attenuate cardiopulmonary disease in rats with neonatal hyperoxia-induced lung injury.Disrupted pulmonary artery cyclic guanosine monophosphate signaling in mice with hyperoxia-induced pulmonary hypertension.Rho-kinase inhibitor Y-27632 attenuates pulmonary hypertension in hyperoxia-exposed newborn rats.Phosphodiesterases: emerging therapeutic targets for neonatal pulmonary hypertension.Current strategies for treating infants with severe bronchopulmonary dysplasia.Pharmacokinetic evaluation of sildenafil as a pulmonary hypertension treatment.Role of reactive oxygen species in neonatal pulmonary vascular disease.Adaptive capacity of the right ventricle: why does it fail?Sildenafil therapy in bronchopulmonary dysplasia-associated pulmonary hypertension: a retrospective study of efficacy and safety.Aberrant cGMP signaling persists during recovery in mice with oxygen-induced pulmonary hypertension.Lung damage analyzed by machine vision on tissue sections of mice.Strategies in early clinical development for the treatment of basic defects of cystic fibrosis.The protective effect of overexpression of extracellular superoxide dismutase on nitric oxide bioavailability in the lung after exposure to hyperoxia stress.Bone Morphogenetic Protein 9 Protects against Neonatal Hyperoxia-Induced Impairment of Alveolarization and Pulmonary Inflammation.Apelin attenuates hyperoxic lung and heart injury in neonatal rats.Isolation of pulmonary artery smooth muscle cells from neonatal mice.Diagnosis and treatment of pulmonary hypertension in infancy.Sildenafil therapy for bronchopulmonary dysplasia: not quite yet.17α-Ethinylestradiol rapidly alters transcript levels of murine coagulation genes via estrogen receptor α.Intraperitoneal injection of cigarette smoke extract induced emphysema, and injury of cardiac and skeletal muscles in BALB/C mice.Beyond pulmonary hypertension: sildenafil for chronic lung disease of prematurity.Human mesenchymal stem cells ameliorate experimental pulmonary hypertension induced by maternal inflammation and neonatal hyperoxia in rats.

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Sildenafil attenuates pulmonary inflammation and fibrin deposition, mortality and right ventricular hypertrophy in neonatal hyperoxic lung injury.

http://www.wikidata.org/entity/Q37190561

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 April 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

@en

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

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type

label

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

@en

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

@nl

prefLabel

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

@en

Sildenafil attenuates pulmonar ...... eonatal hyperoxic lung injury.

@nl

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Respiratory Research

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Sildenafil attenuates pulmonar ...... neonatal hyperoxic lung injury

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Arnoud van der Laarse

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El Houari Laghmani

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Gerry Tm Wagenaar

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Hester Boersma

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Yvonne P de Visser

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S-nitrosothiol signaling in respiratory biology.

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A new mathematical model for relative quantification in real-time RT-PCR

Cyclic nucleotide phosphodiesterase (PDE) superfamily: A new target for the development of specific therapeutic agents

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Keynote review: phosphodiesterase-4 as a therapeutic target

FGFR-3 and FGFR-4 function cooperatively to direct alveogenesis in the murine lung

Vascular endothelial growth factor gene therapy increases survival, promotes lung angiogenesis, and prevents alveolar damage in hyperoxia-induced lung injury: evidence that angiogenesis participates in alveolarization

Phosphodiesterase type 5 is highly expressed in the hypertrophied human right ventricle, and acute inhibition of phosphodiesterase type 5 improves contractility

Mechanisms initiating lung injury in the preterm.

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