Rotavirus antagonizes cellular antiviral responses by inhibiting the nuclear accumulation of STAT1, STAT2, and NF-kappaB. - Wikidata - wikimedia - TriplyDB

Rotavirus antagonizes cellular antiviral responses by inhibiting the nuclear accumulation of STAT1, STAT2, and NF-kappaB.

Rotavirus antagonizes cellular antiviral responses by inhibiting the nuclear accumulation of STAT1, STAT2, and NF-kappaB.

http://www.wikidata.org/entity/Q37192229

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Rotavirus nonstructural protein 1 antagonizes innate immune response by interacting with retinoic acid inducible gene I Rotavirus nonstructural protein 1 suppresses virus-induced cellular apoptosis to facilitate viral growth by activating the cell survival pathways during early stages of infection Inflammatory and oxidative stress in rotavirus infection The battle between rotavirus and its host for control of the interferon signaling pathway Cyclosporin a inhibits rotavirus replication and restores interferon-beta signaling pathway in vitro and in vivo Recombinant human parainfluenza virus type 2 with mutations in V that permit cellular interferon signaling are not attenuated in non-human primates.Porcine reproductive and respiratory syndrome virus inhibits type I interferon signaling by blocking STAT1/STAT2 nuclear translocation.Identification of cellular calcium binding protein calmodulin as a regulator of rotavirus A infection during comparative proteomic study.Characterization of rotavirus RNAs that activate innate immune signaling through the RIG-I-like receptors.Active participation of cellular chaperone Hsp90 in regulating the function of rotavirus nonstructural protein 3 (NSP3).Rotavirus antagonism of the innate immune response The role of type I interferons in intestinal infection, homeostasis, and inflammation.DNA-dependent activator of interferon-regulatory factors inhibits hepatitis B virus replication Rotavirus immune responses and correlates of protection.Release of severe acute respiratory syndrome coronavirus nuclear import block enhances host transcription in human lung cells.Silencing the alarms: Innate immune antagonism by rotavirus NSP1 and VP3 Rotavirus NSP1 mediates degradation of interferon regulatory factors through targeting of the dimerization domain Rotavirus NSP1 protein inhibits interferon-mediated STAT1 activation.Rotavirus and reovirus modulation of the interferon response.Inhibition of rotavirus ECwt infection in ICR suckling mice by N-acetylcysteine, peroxisome proliferator-activated receptor gamma agonists and cyclooxygenase-2 inhibitors.Viral interference with innate immunity by preventing NF-κB activity.Intrinsic and extrinsic negative regulators of nuclear protein transport processes.Viral subversion of nucleocytoplasmic trafficking The Antiviral Effect of Baicalin on Enterovirus 71 In Vitro.Role of cellular caspases, nuclear factor-kappa B and interferon regulatory factors in Bluetongue virus infection and cell fate Rotavirus infection activates the UPR but modulates its activity.Rotavirus replication requires a functional proteasome for effective assembly of viroplasms Reovirus inhibits interferon production by sequestering IRF3 into viral factories.Selective inhibition of host cell signaling for rotavirus antivirals: PI3K/Akt/mTOR-mediated rotavirus pathogenesis.Down the rabbit hole: Is necroptosis truly an innate response to infection?Rotavirus differentially infects and polyclonally stimulates human B cells depending on their differentiation state and tissue of origin.Differences of Rotavirus Vaccine Effectiveness by Country: Likely Causes and Contributing Factors.Rotavirus degrades multiple type interferon receptors to inhibit IFN signaling and protects against mortality from endotoxin in suckling mice.MHC class I expression in intestinal cells is reduced by rotavirus infection and increased in bystander cells lacking rotavirus antigen.Cytoplasmic re-localization and colocalization with viroplasms of host cell proteins, and their role in rotavirus infection.Basal interferon signaling and therapeutic use of interferons in controlling rotavirus infection in human intestinal cells and organoids.

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P2860

Rotavirus antagonizes cellular antiviral responses by inhibiting the nuclear accumulation of STAT1, STAT2, and NF-kappaB.

http://www.wikidata.org/entity/Q37192229

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article científic

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bilimsel makale

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scientific article published on 25 February 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Rotavirus antagonizes cellular ...... f STAT1, STAT2, and NF-kappaB.

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Rotavirus antagonizes cellular ...... f STAT1, STAT2, and NF-kappaB.

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Rotavirus antagonizes cellular ...... f STAT1, STAT2, and NF-kappaB.

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Rotavirus antagonizes cellular ...... f STAT1, STAT2, and NF-kappaB.

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Journal of Virology

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Rotavirus antagonizes cellular ...... f STAT1, STAT2, and NF-kappaB.

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Barbara S Coulson

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Gavan Holloway

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Thanhmai T Truong

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P2860

NF-{kappa}B is transported into the nucleus by importin {alpha}3 and importin {alpha}4

Rotavirus nonstructural protein 1 subverts innate immune response by inducing degradation of IFN regulatory factor 3

Rotavirus NSP1 inhibits expression of type I interferon by antagonizing the function of interferon regulatory factors IRF3, IRF5, and IRF7

Zinc-binding domain of rotavirus NSP1 is required for proteasome-dependent degradation of IRF3 and autoregulatory NSP1 stability

Missing pieces in the NF-kappaB puzzle

Interferons and viruses: an interplay between induction, signalling, antiviral responses and virus countermeasures

Inhibition of interferon signaling by the New York 99 strain and Kunjin subtype of West Nile virus involves blockage of STAT1 and STAT2 activation by nonstructural proteins

Rotavirus infection of MA104 cells is inhibited by Ricinus lectin and separately expressed single binding domains.

Functional classification of interferon-stimulated genes identified using microarrays.

Immune response of children who develop persistent diarrhea following rotavirus infection

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4942-4951

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scholarly article

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10.1128/JVI.01450-08

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10

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83

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2009-02-25T00:00:00Z

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2682104

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