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A single high dose of dexamethasone affects the phosphorylation state of glutamate AMPA receptors in the human limbic systemGluA1 subunit ubiquitination mediates amyloid-β-induced loss of surface α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptorsThe prion protein regulates glutamate-mediated Ca2+ entry and mitochondrial Ca2+ accumulation in neurons.Ketamine and its metabolite (2R,6R)-hydroxynorketamine induce lasting alterations in glutamatergic synaptic plasticity in the mesolimbic circuit.Neuronal pathophysiology featuring PrPC and its control over Ca2+ metabolism.Wnt Signaling Mediates LTP-Dependent Spine Plasticity and AMPAR Localization through Frizzled-7 Receptors.AMPA receptor trafficking and its role in heterosynaptic plasticity.
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description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on August 2016
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Extensive phosphorylation of AMPA receptors in neurons
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Extensive phosphorylation of AMPA receptors in neurons.
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type
label
Extensive phosphorylation of AMPA receptors in neurons
@en
Extensive phosphorylation of AMPA receptors in neurons.
@nl
prefLabel
Extensive phosphorylation of AMPA receptors in neurons
@en
Extensive phosphorylation of AMPA receptors in neurons.
@nl
P2093
P2860
P356
P1476
Extensive phosphorylation of AMPA receptors in neurons
@en
P2093
Graham H Diering
Natasha K Hussain
Richard L Huganir
P2860
P304
P356
10.1073/PNAS.1610631113
P407
P50
P577
2016-08-01T00:00:00Z