Thrombospondin-1 modulates VEGF-A-mediated Akt signaling and capillary survival in the developing retina. - Wikidata - wikimedia - TriplyDB

Thrombospondin-1 modulates VEGF-A-mediated Akt signaling and capillary survival in the developing retina.

Thrombospondin-1 modulates VEGF-A-mediated Akt signaling and capillary survival in the developing retina.

http://www.wikidata.org/entity/Q37200250

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MULTIMERIN2 impairs tumor angiogenesis and growth by interfering with VEGF-A/VEGFR2 pathway CD36 protein is involved in store-operated calcium flux, phospholipase A2 activation, and production of prostaglandin E2 Neonatal systemic inflammation in rats alters retinal vessel development and simulates pathologic features of retinopathy of prematurity.Thrombospondin-1 inhibits VEGF receptor-2 signaling by disrupting its association with CD47.Analysis of candidate genes for macular telangiectasia type 2 Chronic delivery of a thrombospondin-1 mimetic decreases skeletal muscle capillarity in mice Priming of the vascular endothelial growth factor signaling pathway by thrombospondin-1, CD36, and spleen tyrosine kinase.Common variants near FRK/COL10A1 and VEGFA are associated with advanced age-related macular degeneration.The thrombospondins.Interaction of thrombospondin1 and CD36 contributes to obesity-associated podocytopathy.The matricellular protein thrombospondin-1 globally regulates cardiovascular function and responses to stress via CD47.Thrombospondin-1 is a novel negative regulator of liver regeneration after partial hepatectomy through transforming growth factor-beta1 activation in mice.Molecular basis for the regulation of angiogenesis by thrombospondin-1 and -2.Thrombospondin-1 modulates VEGF signaling via CD36 by recruiting SHP-1 to VEGFR2 complex in microvascular endothelial cells Thrombospondins in the heart: potential functions in cardiac remodeling.Thrombospondin-1 as a Paradigm for the Development of Antiangiogenic Agents Endowed with Multiple Mechanisms of Action.Negative regulators of angiogenesis: important targets for treatment of exudative AMD.Tetrahydrobiopterin (BH4) deficiency is associated with augmented inflammation and microvascular degeneration in the retina.Thrombospondin-1 modulates vascular endothelial growth factor activity at the receptor level.Integration of pro- and anti-angiogenic signals by endothelial cells.Alternative transcription of a shorter, non-anti-angiogenic thrombospondin-2 variant in cancer-associated blood vessels.

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Thrombospondin-1 modulates VEGF-A-mediated Akt signaling and capillary survival in the developing retina.

http://www.wikidata.org/entity/Q37200250

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@en

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@nl

type

label

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@en

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@nl

prefLabel

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@en

Thrombospondin-1 modulates VEG ...... ival in the developing retina.

@nl

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AJPHEART.01246.2008

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American Journal of Physiology - Heart and Circulatory Physiology

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Thrombospondin-1 modulates VEG ...... vival in the developing retina

@en

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Benjamin D Hopkins

http://www.w3.org/2001/XMLSchema#string

Carole Perruzzi

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Irit Adini

http://www.w3.org/2001/XMLSchema#string

Jack Lawler

http://www.w3.org/2001/XMLSchema#string

Jingfang Sun

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Kaoru Tsujikawa

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Laura E Benjamin

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Paul Bornstein

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Thrombospondin-1 inhibits nitric oxide signaling via CD36 by inhibiting myristic acid uptake

Thrombospondin-1

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

Membrane glycoprotein IV (CD36) is physically associated with the Fyn, Lyn, and Yes protein-tyrosine kinases in human platelets

Thrombospondins use the VLDL receptor and a nonapoptotic pathway to inhibit cell division in microvascular endothelial cells

Thrombospondin 1 and type I repeat peptides of thrombospondin 1 specifically induce apoptosis of endothelial cells

Src-related protein tyrosine kinases are physically associated with the surface antigen CD36 in human dermal microvascular endothelial cells

Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis

The antiangiogenic effect of thrombospondin-2 is mediated by CD36 and modulated by histidine-rich glycoprotein

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scholarly article

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10.1152/AJPHEART.01246.2008

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5

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296

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Robert Swerlick

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2009-03-20T00:00:00Z

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24218366

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19304944

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