Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence. - Wikidata - wikimedia - TriplyDB

Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence.

Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence.

http://www.wikidata.org/entity/Q37202938

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Leishmania carbon metabolism in the macrophage phagolysosome- feast or famine?Control of macrophage metabolism and activation by mTOR and Akt signaling Intraspecies competition for niches in the distal gut dictate transmission during persistent Salmonella infection Replication of Salmonella enterica Serovar Typhimurium in Human Monocyte-Derived Macrophages TH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Bacterial recognition pathways that lead to inflammasome activation Exploiting host immunity: the Salmonella paradigm Host Delivery of Favorite Meals for Intracellular Pathogens The role of macrophage polarization in infectious and inflammatory diseases Malaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function.Macrophages in homeostatic immune function.Polarizing the T helper 17 response in Citrobacter rodentium infection via expression of resistin-like molecule α.Salmonella acquires ferrous iron from haemophagocytic macrophages Salmonella enterica infection stimulates macrophages to hemophagocytose Increased ferroportin-1 expression and rapid splenic iron loss occur with anemia caused by Salmonella enterica Serovar Typhimurium infection in mice Role of peroxisome proliferator-activated receptors gene polymorphisms in type 2 diabetes and metabolic syndrome.The role of autophagy in intracellular pathogen nutrient acquisition.In Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible Mice A Role for the Non-Canonical Wnt-β-Catenin and TGF-β Signaling Pathways in the Induction of Tolerance during the Establishment of a Salmonella enterica Serovar Enteritidis Persistent Cecal Infection in Chickens Macrophage cell death upon intracellular bacterial infection.Disruption of glycolytic flux is a signal for inflammasome signaling and pyroptotic cell death Infection Susceptibility in Gastric Intrinsic Factor (Vitamin B12)-Defective Mice Is Subject to Maternal Influences Bacterial Metabolism Shapes the Host-Pathogen Interface Immunometabolic Phenotype Alterations Associated with the Induction of Disease Tolerance and Persistent Asymptomatic Infection of Salmonella in the Chicken Intestine.Immune Homeostatic Macrophages Programmed by the Bacterial Surface Protein NhhA Potentiate Nasopharyngeal Carriage of Neisseria meningitidis.Intracellular Salmonella metabolism.AMPK and mTOR: sensors and regulators of immunometabolic changes during Salmonella infection in the chicken.Brucella abortus Induces a Warburg Shift in Host Metabolism That Is Linked to Enhanced Intracellular Survival of the Pathogen.Metabolic crosstalk between host and pathogen: sensing, adapting and competing.Chronic Bacterial Pathogens: Mechanisms of Persistence.Salmonella and the Inflammasome: Battle for Intracellular Dominance.Disseminated infections with antibiotic-resistant non-typhoidal Salmonella strains: contributions of host and pathogen factors.Single-cell RNA-seq ties macrophage polarization to growth rate of intracellular Salmonella.Interleukin-10 Production by T and B Cells Is a Key Factor to Promote Systemic Salmonella enterica Serovar Typhimurium Infection in Mice.To Eat and to Be Eaten: Mutual Metabolic Adaptations of Immune Cells and Intracellular Bacterial Pathogens upon Infection.The infectious intracellular lifestyle of Salmonella enterica relies on the adaptation to nutritional conditions within the Salmonella-containing vacuole.Salmonella typhimurium-induced M1 macrophage polarization is dependent on the bacterial O antigen.Ribosome Rescue Inhibitors Kill Actively Growing and Nonreplicating Persister Mycobacterium tuberculosis Cells.The metabolic enzyme fructose-1,6-bisphosphate aldolase acts as a transcriptional regulator in pathogenic Francisella.Utilization of host polyamines in alternatively activated macrophages promotes chronic infection by Brucella abortus.

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Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence.

http://www.wikidata.org/entity/Q37202938

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on August 2013

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vedecký článok

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vetenskaplig artikel

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vědecký článek

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name

Salmonella require the fatty a ...... ial for long-term persistence.

@en

Salmonella require the fatty a ...... ial for long-term persistence.

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type

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Salmonella require the fatty a ...... ial for long-term persistence.

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Salmonella require the fatty a ...... ial for long-term persistence.

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Salmonella require the fatty a ...... ial for long-term persistence.

@en

Salmonella require the fatty a ...... ial for long-term persistence.

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J.CHOM.2013.07.010

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Cell Host & Microbe

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Salmonella require the fatty a ...... ial for long-term persistence.

@en

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Ajay Chawla

http://www.w3.org/2001/XMLSchema#string

Amanda Jacobson

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Denise M Monack

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Jeffery S Cox

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Lata Mukundan

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Lilian Lam

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Nicholas A Eisele

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Paolo S Manzanillo

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Thomas Ruby

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P2860

Molecular recognition of fatty acids by peroxisome proliferator-activated receptors

PPAR delta: a dagger in the heart of the metabolic syndrome.

PPAR-delta senses and orchestrates clearance of apoptotic cells to promote tolerance

Exploring the full spectrum of macrophage activation

Hemophagocytic macrophages harbor Salmonella enterica during persistent infection

Systems approach to investigating host-pathogen interactions in infections with the biothreat agent Francisella. Constraints-based model of Francisella tularensis

Alternative activation of macrophages

Persistence of Mycobacterium tuberculosis in macrophages and mice requires the glyoxylate shunt enzyme isocitrate lyase

Mycobacterial persistence requires the utilization of host cholesterol

Interleukin-4-dependent production of PPAR-gamma ligands in macrophages by 12/15-lipoxygenase

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171-182

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scholarly article

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10.1016/J.CHOM.2013.07.010

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2

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14

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2013-08-01T00:00:00Z

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23954156

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