Simvastatin up-regulates adenosine deaminase and suppresses osteopontin expression in COPD patients through an IL-13-dependent mechanism

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Association of the IL-13 polymorphisms rs1800925 and rs20541 with chronic obstructive pulmonary disease risk: An updated meta-analysis.

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Simvastatin up-regulates adenosine deaminase and suppresses osteopontin expression in COPD patients through an IL-13-dependent mechanism

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bilimsel makale

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scientific article published on 24 August 2016

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Simvastatin up-regulates adeno ...... h an IL-13-dependent mechanism

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Simvastatin up-regulates adeno ...... an IL-13-dependent mechanism.

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Simvastatin up-regulates adeno ...... h an IL-13-dependent mechanism

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Simvastatin up-regulates adeno ...... an IL-13-dependent mechanism.

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Simvastatin up-regulates adeno ...... h an IL-13-dependent mechanism

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Simvastatin up-regulates adeno ...... an IL-13-dependent mechanism.

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Simvastatin up-regulates adeno ...... h an IL-13-dependent mechanism

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Adisak Wongkajornsilp

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Kanda Kasetsinsombat

http://www.w3.org/2001/XMLSchema#string

Kittipong Maneechotesuwan

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Global Strategy for the Diagnosis, Management, and Prevention of Chronic Obstructive Pulmonary Disease

A distinctive alveolar macrophage activation state induced by cigarette smoking

Adenosine receptors: therapeutic aspects for inflammatory and immune diseases.

Respiratory syncytial virus infections enhance cigarette smoke induced COPD in mice

Adenosine-activated mast cells induce IgE synthesis by B lymphocytes: an A2B-mediated process involving Th2 cytokines IL-4 and IL-13 with implications for asthma

NK-104, a 3-hydroxy-3-methylglutaryl coenzyme A reductase inhibitor, reduces osteopontin expression by rat aortic smooth muscle cells

Metabolic consequences of adenosine deaminase deficiency in mice are associated with defects in alveogenesis, pulmonary inflammation, and airway obstruction

Alterations in adenosine metabolism and signaling in patients with chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis

Lung volumes and forced ventilatory flows. Report Working Party Standardization of Lung Function Tests, European Community for Steel and Coal. Official Statement of the European Respiratory Society

Adenosine and osteopontin contribute to the development of chronic obstructive pulmonary disease.

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s12931-016-0424-6

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scholarly article

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10.1186/S12931-016-0424-6

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Peter J. Barnes

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2016-08-24T00:00:00Z

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1043703108

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27557561

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chronic obstructive pulmonary disease

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4997725

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Simvastatin up-regulates adenosine deaminase and suppresses osteopontin expression in COPD patients through an IL-13-dependent mechanism

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Simvastatin up-regulates adenosine deaminase and suppresses osteopontin expression in COPD patients through an IL-13-dependent mechanism

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