Elevated expression of caspase-3 inhibitors, survivin and xIAP correlates with low levels of apoptosis in active rheumatoid synovium. - Wikidata - wikimedia - TriplyDB

Elevated expression of caspase-3 inhibitors, survivin and xIAP correlates with low levels of apoptosis in active rheumatoid synovium.

Elevated expression of caspase-3 inhibitors, survivin and xIAP correlates with low levels of apoptosis in active rheumatoid synovium.

http://www.wikidata.org/entity/Q37207081

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Gadd45beta deficiency in rheumatoid arthritis: enhanced synovitis through JNK signaling.HIV infection enhances TRAIL-induced cell death in macrophage by down-regulating decoy receptor expression and generation of reactive oxygen species.The oncofetal gene survivin is re-expressed in osteoarthritis and is required for chondrocyte proliferation in vitro Activation of Fms-like tyrosine kinase 3 signaling enhances survivin expression in a mouse model of rheumatoid arthritis TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis.The proto-oncogene survivin splice variant 2B is induced by PDGF and leads to cell proliferation in rheumatoid arthritis fibroblast-like synoviocytes.The X-Linked Inhibitor of Apoptosis Protein Inhibitor Embelin Suppresses Inflammation and Bone Erosion in Collagen Antibody Induced Arthritis Mice Suppressed diversity of survivin splicing in active rheumatoid arthritis.Intracellular Signaling Pathways in Rheumatoid Arthritis Th17 and Th22 cells in psoriatic arthritis and psoriasis Smoking in combination with antibodies to cyclic citrullinated peptides is associated with persistently high levels of survivin in early rheumatoid arthritis: a prospective cohort study.Survivin but not Fms-like tyrosine kinase 3 ligand is up-regulated before the onset of rheumatoid arthritis: a pilot study.TNF-related apoptosis-inducing ligand (TRAIL) in rheumatoid arthritis: what's new?Negative Regulators of JAK/STAT Signaling in Rheumatoid Arthritis and Osteoarthritis.Down-regulation of microRNA-34a* in rheumatoid arthritis synovial fibroblasts promotes apoptosis resistance.Survivin co-ordinates formation of follicular T-cells acting in synergy with Bcl-6.Intracellular apoptotic pathways: a potential target for reducing joint damage in rheumatoid arthritis.Anti-interleukin-6 signalling therapy rebalances the disrupted cytokine production of B cells from patients with active rheumatoid arthritis.Apoptosis in the rheumatoid arthritis synovial membrane: modulation by disease-modifying anti-rheumatic drug treatment.CTGF promotes articular damage by increased proliferation of fibroblast-like synoviocytes in rheumatoid arthritis.Lysophosphatidic acid receptor 1 suppression sensitizes rheumatoid fibroblast-like synoviocytes to tumor necrosis factor-induced apoptosis.Higher incidence of sperm granuloma in the epididymis of C57BL/6N mice.Survivin Measurement improves Clinical Prediction of Transition From Arthralgia to RA-Biomarkers to Improve Clinical Sensitivity of Transition From Arthralgia to RA Reduced apoptosis correlates with enhanced autophagy in synovial tissues of rheumatoid arthritis

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Elevated expression of caspase-3 inhibitors, survivin and xIAP correlates with low levels of apoptosis in active rheumatoid synovium.

http://www.wikidata.org/entity/Q37207081

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 January 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Elevated expression of caspase ...... in active rheumatoid synovium.

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Arthritis Research and Therapy

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Elevated expression of caspase ...... in active rheumatoid synovium.

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Anak A S S K Dharmapatni

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Andreas Evdokiou

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Christopher A Holding

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David M Findlay

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David R Haynes

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Helen Weedon

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Malcolm D Smith

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Michael J Ahern

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Paul Chen

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Xiao N Xu

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P2860

X-linked IAP is a direct inhibitor of cell-death proteases

The novel receptor TRAIL-R4 induces NF-kappaB and protects against TRAIL-mediated apoptosis, yet retains an incomplete death domain

Balance between survivin, a key member of the apoptosis inhibitor family, and its specific antibodies determines erosivity in rheumatoid arthritis

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Structural basis of IAP recognition by Smac/DIABLO

The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis

Differential localization and regulation of death and decoy receptors for TNF-related apoptosis-inducing ligand (TRAIL) in human melanoma cells

Control of TRAIL-induced apoptosis by a family of signaling and decoy receptors

A novel receptor for Apo2L/TRAIL contains a truncated death domain

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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scholarly article

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10.1186/AR2603

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1

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11

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Gavin R Screaton

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2009-01-27T00:00:00Z

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apoptotic process

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2688245

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