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Nucleosome Dancing at the Tempo of Histone Tail AcetylationFunctional redundancy of SWI/SNF catalytic subunits in maintaining vascular endothelial cells in the adult heartThe SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell ProliferationTranscriptional Gene Silencing (TGS) via the RNAi Machinery in HIV-1 InfectionsThe histone-fold complex MHF is remodeled by FANCM to recognize branched DNA and protect genome stability.Expanding the phenotypic spectrum of ARID1B-mediated disorders and identification of altered cell-cycle dynamics due to ARID1B haploinsufficiency.Mechanisms of HIV Transcriptional Regulation by Drugs of Abuse.Varying modulation of HIV-1 LTR activity by Baf complexes.SWI/SNF chromatin-remodeling enzymes Brahma-related gene 1 (BRG1) and Brahma (BRM) are dispensable in multiple models of postnatal angiogenesis but are required for vascular integrity in infant mice.Sequence-based prediction of single nucleosome positioning and genome-wide nucleosome occupancyHuman immunodeficiency virus (HIV) latency: the major hurdle in HIV eradicationThe effects of cocaine on HIV transcriptionSwi/Snf chromatin remodeling/tumor suppressor complex establishes nucleosome occupancy at target promotersHistone H3 phosphorylation near the nucleosome dyad alters chromatin structure.HIV latency.Swi2/Snf2 remodelers: hybrid views on hybrid molecular machines.Epithelial stem cells in adult skin.ARID1B-mediated disorders: Mutations and possible mechanisms.Characterization of a Brg1 hypomorphic allele demonstrates that genetic and biochemical activity are tightly correlated.SWI/SNF- and RSC-catalyzed nucleosome mobilization requires internal DNA loop translocation within nucleosomes.The divalent cations Ca2+ and Mg2+ play specific roles in stabilizing histone-DNA interactions within nucleosomes that are partially redundant with the core histone tail domains.Combined gene dosage requirement for SWI/SNF catalytic subunits during early mammalian development.A comprehensive molecular study on Coffin-Siris and Nicolaides-Baraitser syndromes identifies a broad molecular and clinical spectrum converging on altered chromatin remodeling.The BRG1 ATPase of human SWI/SNF chromatin remodeling enzymes as a driver of cancer.
P2860
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P2860
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 October 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mechanism(s) of SWI/SNF-induced nucleosome mobilization
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type
label
Mechanism(s) of SWI/SNF-induced nucleosome mobilization
@en
prefLabel
Mechanism(s) of SWI/SNF-induced nucleosome mobilization
@en
P2093
P2860
P356
P1433
P1476
Mechanism(s) of SWI/SNF-induced nucleosome mobilization
@en
P2093
Angela Balliano
Jeffrey J Hayes
P2860
P304
P356
10.1002/CBIC.201000455
P577
2010-10-28T00:00:00Z