Axons are injured by antigen-specific CD8(+) T cells through a MHC class I- and granzyme B-dependent mechanism.
about
Major Histocompatibility Complex I Expression by Motor Neurons and Its Implication in Amyotrophic Lateral SclerosisCD8(+) T-Cells as Immune Regulators of Multiple SclerosisAntigen Presentation, Autoantigens, and Immune Regulation in Multiple Sclerosis and Other Autoimmune DiseasesMultiple Sclerosis and T Lymphocytes: An Entangled StoryCharacteristics and critical function of CD8+ T cells in the Toxoplasma-infected brain.Neuronal MHC-I expression and its implications in synaptic function, axonal regeneration and Parkinson's and other brain diseasesType 1 narcolepsy: a CD8(+) T cell-mediated disease?Axonal degeneration in multiple sclerosis: defining therapeutic targets by identifying the causes of pathology.Oligodendrocytes: Functioning in a Delicate Balance Between High Metabolic Requirements and Oxidative Damage.Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathyInflammatory cytokine-induced changes in neural network activity measured by waveform analysis of high-content calcium imaging in murine cortical neurons.Microfluidic platforms for the study of neuronal injury in vitro.Retrograde interferon-gamma signaling induces major histocompatibility class I expression in human-induced pluripotent stem cell-derived neurons.Prior regular exercise improves clinical outcome and reduces demyelination and axonal injury in experimental autoimmune encephalomyelitis.A Novel Association of Polymorphism in the ITGA4 Gene Encoding the VLA-4 α4 Subunit with Increased Risk of Alzheimer's Disease.
P2860
Q26744608-D81D3B78-BDE6-4FDB-B82D-E53126B5A2C3Q26772339-1A56DDA2-2825-48DE-8553-BD46DF0E4579Q28080791-8ED0DE56-6389-4575-A1D5-7D83868FF4E3Q28086853-7D1139D0-5F33-4E39-A787-97A844A4A3A5Q30354096-52800D53-7B6D-4AD9-9751-82617B292D5BQ38263500-F47FF6FC-9DA5-4444-9BAE-235ED6109EE3Q38533728-43065D41-7C67-4772-B2BA-2B4234619A81Q38653397-88A6BCB8-7512-40C9-8386-329921609204Q38974735-31F1D380-6370-4343-8DB3-1014D5D9789AQ39210948-9A4F70DB-421E-4001-A91E-E32D02C6844FQ40074599-37700892-A757-441E-97AC-94097A26A29EQ47240527-AB6DBFDB-5CB7-4448-826A-00146DB3F7F4Q50282106-43788178-B190-4AA9-9D07-AFD56BE1E4A2Q50985177-C7250BC5-19AD-40B5-8168-AAC32C90AD42Q55259831-4FA621B2-4933-4F77-A652-91F1D0E52B85
P2860
Axons are injured by antigen-specific CD8(+) T cells through a MHC class I- and granzyme B-dependent mechanism.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 27 July 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Axons are injured by antigen-specific CD8
@nl
Axons are injured by antigen-s ...... ranzyme B-dependent mechanism.
@en
type
label
Axons are injured by antigen-specific CD8
@nl
Axons are injured by antigen-s ...... ranzyme B-dependent mechanism.
@en
prefLabel
Axons are injured by antigen-specific CD8
@nl
Axons are injured by antigen-s ...... ranzyme B-dependent mechanism.
@en
P2093
P2860
P1476
Axons are injured by antigen-s ...... ranzyme B-dependent mechanism.
@en
P2093
Brian M Sauer
Charles L Howe
William F Schmalstieg
P2860
P304
P356
10.1016/J.NBD.2013.07.010
P577
2013-07-27T00:00:00Z