Loss of TIMP3 enhances interstitial nephritis and fibrosis. - Wikidata - wikimedia - TriplyDB

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

http://www.wikidata.org/entity/Q37211116

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Diverse functions of matrix metalloproteinases during fibrosis Tubulointerstitial injury and the progression of chronic kidney disease Matrix metalloproteinases in kidney homeostasis and diseases Diabetic kidney disease: from physiology to therapeutics Characterization of angiotensin-converting enzyme 2 ectodomain shedding from mouse proximal tubular cells Down-regulation of TIMP3 leads to increase in TACE expression and TNFα production by placental trophoblast cells.ADAM-17: the enzyme that does it all.A disintegrin and metalloenzyme (ADAM) 17 activation is regulated by α5β1 integrin in kidney mesangial cells.Loss of Timp3 gene leads to abdominal aortic aneurysm formation in response to angiotensin II A glimpse of matrix metalloproteinases in diabetic nephropathy.Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44.Fibrosis and progression of autosomal dominant polycystic kidney disease (ADPKD).Absence of nicotinic acetylcholine receptor α7 subunit amplifies inflammation and accelerates onset of fibrosis: an inflammatory kidney model.Inhibition of Thrombin-Activated Fibrinolysis Inhibitor Increases Survival in Experimental Kidney Fibrosis.Pericyte TIMP3 and ADAMTS1 modulate vascular stability after kidney injury.The history of matrix metalloproteinases: milestones, myths, and misperceptions.Cellular mechanisms of tissue fibrosis. 3. Novel mechanisms of kidney fibrosis.Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Accelerates the Progression of Renal Fibrosis in Lupus Nephritis by Activating SMAD and p38 MAPK in TGF-β1 Signaling Pathway ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice.Renal studies provide an insight into cardiac extracellular matrix remodeling during health and disease.Inhibition of the TGFβ signalling pathway by cGMP and cGMP-dependent kinase I in renal fibrosis.Tissue inhibitor of metalloproteinases (TIMPs) in heart failure.Rethinking glomerular basement membrane thickening in diabetic nephropathy: adaptive or pathogenic?Regulation of TIMP3 in diabetic nephropathy: a role for microRNAs.Loss of TIMP3 underlies diabetic nephropathy via FoxO1/STAT1 interplay.Functional differences of miR-125b on the invasion of primary glioblastoma CD133-negative cells and CD133-positive cells.Human recombinant ACE2 reduces the progression of diabetic nephropathy.Loss of TIMP3 selectively exacerbates diabetic nephropathy.Proteomic and metabolomic characterization of streptozotocin-induced diabetic nephropathy in TIMP3-deficient mice.Differential expression and localization of human tissue inhibitors of metalloproteinases in proliferative diabetic retinopathy.TIMP3 deficiency exacerbates iron-overload mediated cardiomyopathy and liver disease.Skeletal muscle TLR4 and TACE are associated with body fat percentage in older adults.Resolution of organ fibrosis.ADAM17 up-regulation in renal transplant dysfunction and non-transplant-related renal fibrosis.Delayed Treatment with Plasminogen Activator Inhibitor-1 Decoys Reduces Tubulointerstitial Fibrosis Targeting enhancer of zeste homolog 2 protects against acute kidney injury

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Loss of TIMP3 enhances interstitial nephritis and fibrosis.

http://www.wikidata.org/entity/Q37211116

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 30 April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@en

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@nl

type

label

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@en

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@nl

prefLabel

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@en

Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@nl

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P1433

Journal of the American Society of Nephrology

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Loss of TIMP3 enhances interstitial nephritis and fibrosis.

@en

P2093

Ahmed Awad

http://www.w3.org/2001/XMLSchema#string

Andrew M Herzenberg

http://www.w3.org/2001/XMLSchema#string

Gavin Y Oudit

http://www.w3.org/2001/XMLSchema#string

James W Scholey

http://www.w3.org/2001/XMLSchema#string

Nobuyo Maeda

http://www.w3.org/2001/XMLSchema#string

Vijay Kandalam

http://www.w3.org/2001/XMLSchema#string

Xiuhua Wang

http://www.w3.org/2001/XMLSchema#string

Xiuhua Ziou

http://www.w3.org/2001/XMLSchema#string

Zamaneh Kassiri

http://www.w3.org/2001/XMLSchema#string

P2860

ADAM 12-S cleaves IGFBP-3 and IGFBP-5 and is inhibited by TIMP-3

Kidney-targeted Birt-Hogg-Dube gene inactivation in a mouse model: Erk1/2 and Akt-mTOR activation, cell hyperproliferation, and polycystic kidneys

Intermolecular autolytic cleavage can contribute to the activation of progelatinase A by cell membranes

Expression of the type IV collagenase system during mouse kidney development and tubule segmentation.

Response by the corpuscles of Stannius to hypotensive stimuli in three divergent ray-finned fishes (Amia calva, Anguilla rostrata, and Catastomus commersoni): cardiovascular and morphological changes.

Molecular basis of renal fibrosis.

Expression analysis of the entire MMP and TIMP gene families during mouse tissue development.

Kidney stones: pathophysiology and medical management.

Obstructive nephropathy and renal fibrosis.

Tubular apoptosis in the pathophysiology of renal disease.

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1223-1235

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scholarly article

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10.1681/ASN.2008050492

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6

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20

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2009-04-30T00:00:00Z

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19406980

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2689897

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