Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis - Wikidata - wikimedia - TriplyDB

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

http://www.wikidata.org/entity/Q37211135

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Invariant natural killer T cell agonist modulates experimental focal and segmental glomerulosclerosis Peripheral blood CD8αα+CD11c+MHC-II+CD3- cells attenuate autoimmune glomerulonephritis in rats.Spontaneous focal activation of invariant natural killer T (iNKT) cells in mouse liver and kidney.Innate-like and conventional T cell populations from hemodialyzed and kidney transplanted patients are equally compromised Asymptomatic autoantibodies associate with future anti-glomerular basement membrane disease Activation of Notch3 in Glomeruli Promotes the Development of Rapidly Progressive Renal Disease Immature renal dendritic cells recruit regulatory CXCR6(+) invariant natural killer T cells to attenuate crescentic GN.Protective effects of genetic inhibition of Discoidin Domain Receptor 1 in experimental renal disease Whole-transcriptome analysis of UUO mouse model of renal fibrosis reveals new molecular players in kidney diseases Advances in human antiglomerular basement membrane disease.Update on crescentic glomerulonephritis.NFκB-Induced Periostin Activates Integrin-β3 Signaling to Promote Renal Injury in GN.The Players: Cells Involved in Glomerular Disease.Redefining lupus nephritis: clinical implications of pathophysiologic subtypes.Administration of α-Galactosylceramide Improves Adenine-Induced Renal Injury An overview of molecular mechanism of nephrotic syndrome.Adriamycin-induced nephropathy in rats: functional and cellular effects characterized by MRI.Alteration of connexin expression is an early signal for chronic kidney disease.Repeated administration of alpha-galactosylceramide ameliorates experimental lupus nephritis in mice.

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Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

http://www.wikidata.org/entity/Q37211135

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

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bilimsel makale

@tr

scientific article published on 21 May 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

@en

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis.

@nl

type

label

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

@en

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis.

@nl

prefLabel

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

@en

Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis.

@nl

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Journal of the American Society of Nephrology

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Invariant natural killer T cells and TGF-beta attenuate anti-GBM glomerulonephritis

@en

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Cédric Rafat

http://www.w3.org/2001/XMLSchema#string

Emmanuel Letavernier

http://www.w3.org/2001/XMLSchema#string

Eric Rondeau

http://www.w3.org/2001/XMLSchema#string

Laurent Mesnard

http://www.w3.org/2001/XMLSchema#string

Yves Tillet

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P2860

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death

RANTES and monocyte chemoattractant protein-1 (MCP-1) play an important role in the inflammatory phase of crescentic nephritis, but only MCP-1 is involved in crescent formation and interstitial fibrosis

NKT cell activation mediates neutrophil IFN-gamma production and renal ischemia-reperfusion injury.

Ultrasound-microbubble-mediated gene transfer of inducible Smad7 blocks transforming growth factor-beta signaling and fibrosis in rat remnant kidney.

Immunopathogenesis of crescentic glomerulonephritis.

Real-time PCR for mRNA quantitation.

Activation of natural killer (NK) T cells during murine cytomegalovirus infection enhances the antiviral response mediated by NK cells

Transforming growth factor-beta controls development, homeostasis, and tolerance of T cells by regulatory T cell-dependent and -independent mechanisms.

T cells in glomerulonephritis.

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1282-1292

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scholarly article

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10.1681/ASN.2008040433

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6

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20

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Alexandre Castro Keller

M C Leite de Moraes

S Vandermeersch

Marie-Laure Michel

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2009-05-21T00:00:00Z

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19470687

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glomerulonephritis

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2689902

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