Overexpression of phosphatase and tensin homolog improves fitness and decreases Plasmodium falciparum development in Anopheles stephensi.

Overexpression of phosphatase and tensin homolog improves fitness and decreases Plasmodium falciparum development in Anopheles stephensi.

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http://www.wikidata.org/entity/Q37211363
Human IGF1 regulates midgut oxidative stress and epithelial homeostasis to balance lifespan and Plasmodium falciparum resistance in Anopheles stephensiEngineered single nucleotide polymorphisms in the mosquito MEK docking site alter Plasmodium berghei development in Anopheles gambiae.Abscisic acid induces a transient shift in signaling that enhances NF-κB-mediated parasite killing in the midgut of Anopheles stephensi without reducing lifespan or fecundity.Two insulin-like peptides differentially regulate malaria parasite infection in the mosquito through effects on intermediary metabolism.Molecular analysis of Aedes aegypti classical protein tyrosine phosphatases uncovers an ortholog of mammalian PTP-1B implicated in the control of egg production in mosquitoes.Knockdown of mitogen-activated protein kinase (MAPK) signalling in the midgut of Anopheles stephensi mosquitoes using antisense morpholinos.Effects of ingested vertebrate-derived factors on insect immune responses.Engineering the control of mosquito-borne infectious diseases.Protein kinase C-dependent signaling controls the midgut epithelial barrier to malaria parasite infection in anopheline mosquitoesIncreased Akt signaling in the mosquito fat body increases adult survivorship.Immune response and insulin signalling alter mosquito feeding behaviour to enhance malaria transmission potential.Plasmodium falciparum suppresses the host immune response by inducing the synthesis of insulin-like peptides (ILPs) in the mosquito Anopheles stephensiAnopheles stephensi p38 MAPK signaling regulates innate immunity and bioenergetics during Plasmodium falciparum infectionSupplementation with Abscisic Acid Reduces Malaria Disease Severity and Parasite Transmission.Infection with Plasmodium berghei ookinetes alters protein expression in the brain of Anopheles albimanus mosquitoes.Host-pathogen interactions in malaria: cross-kingdom signaling and mitochondrial regulation.Gene Drive for Mosquito Control: Where Did It Come from and Where Are We Headed?
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Overexpression of phosphatase and tensin homolog improves fitness and decreases Plasmodium falciparum development in Anopheles stephensi.

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http://www.wikidata.org/entity/Q37211363
description
article científic
@ca
article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 June 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Overexpression of phosphatase ...... opment in Anopheles stephensi.
@en
Overexpression of phosphatase ...... opment in Anopheles stephensi.
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type
label
Overexpression of phosphatase ...... opment in Anopheles stephensi.
@en
Overexpression of phosphatase ...... opment in Anopheles stephensi.
@nl
prefLabel
Overexpression of phosphatase ...... opment in Anopheles stephensi.
@en
Overexpression of phosphatase ...... opment in Anopheles stephensi.
@nl
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Overexpression of phosphatase ...... opment in Anopheles stephensi.
@en
P2093
P2860
P304
P31
P356
10.1016/J.MICINF.2013.05.006
P433
P478
P577
P698
P921
P932