Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease. - Wikidata - wikimedia - TriplyDB

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

http://www.wikidata.org/entity/Q37211903

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Inflammation and intracellular metabolism: new targets in OA Emerging regulators of the inflammatory process in osteoarthritis Osteoarthritis year 2011 in review: biology Nutraceuticals: potential for chondroprotection and molecular targeting of osteoarthritis The age-related changes in cartilage and osteoarthritis The beneficial effects of exercise on cartilage are lost in mice with reduced levels of ECSOD in tissues Procainamide Inhibits DNA Methylation and Alleviates Multiple Organ Dysfunction in Rats with Endotoxic Shock Increased DNA Methylation and Reduced Expression of Transcription Factors in Human Osteoarthritis Cartilage.Bach1 deficiency reduces severity of osteoarthritis through upregulation of heme oxygenase-1 Immunopathogenesis of osteoarthritis Identification of new autoantibody specificities directed at proteins involved in the transforming growth factor β pathway in patients with systemic sclerosis Proteome analysis during chondrocyte differentiation in a new chondrogenesis model using human umbilical cord stroma mesenchymal stem cells.Mitochondrial DNA (mtDNA) haplogroups and serum levels of anti-oxidant enzymes in patients with osteoarthritis.Superoxide dismutase 1 loss disturbs intracellular redox signaling, resulting in global age-related pathological changes.Association of reduced type IX collagen gene expression in human osteoarthritic chondrocytes with epigenetic silencing by DNA hypermethylation FoxO transcription factors support oxidative stress resistance in human chondrocytes Peroxisome proliferator-activated receptor γ coactivator 1α and FoxO3A mediate chondroprotection by AMP-activated protein kinase.Mitochondrial respiration and redox coupling in articular chondrocytes Eupatilin Exerts Antinociceptive and Chondroprotective Properties in a Rat Model of Osteoarthritis by Downregulating Oxidative Damage and Catabolic Activity in Chondrocytes Mechanical overloading causes mitochondrial superoxide and SOD2 imbalance in chondrocytes resulting in cartilage degeneration.Redox signalling and mitochondrial stress responses; lessons from inborn errors of metabolism.Effects of aging on articular cartilage homeostasis.Synovium and the innate inflammatory network in osteoarthritis progression.Delphinidin inhibits IL-1β-induced activation of NF-κB by modulating the phosphorylation of IRAK-1(Ser376) in human articular chondrocytes.Therapeutic effects of the superoxide dismutase mimetic compound MnIIMe2DO2A on experimental articular pain in rats.Cytosolic caspases mediate mislocalised SOD2 depletion in an in vitro model of chronic prion infection.Ageing and the pathogenesis of osteoarthritis.The future of osteoarthritis therapeutics: targeted pharmacological therapy Dysregulated FOXO transcription factors in articular cartilage in aging and osteoarthritis.Ursodeoxycholic Acid ameliorates pain severity and cartilage degeneration in monosodium iodoacetate-induced osteoarthritis in rats.Impact of inflammation on the osteoarthritic niche: implications for regenerative medicine.Epigenetics in osteoarthritis and its implication for future therapeutics.Changes in the epigenetic status of the SOX-9 promoter in human osteoarthritic cartilage.Epigenetics of osteoarticular diseases: recent developments.Oxidative stress and chronic inflammation in osteoarthritis: can NRF2 counteract these partners in crime?ELF3 modulates type II collagen gene (COL2A1) transcription in chondrocytes by inhibiting SOX9-CBP/p300-driven histone acetyltransferase activity.Global and targeted metabolomics of synovial fluid discovers special osteoarthritis metabolites.Computational modelling folate metabolism and DNA methylation: implications for understanding health and ageing.Quantitative Mass Spectrometry To Study Inflammatory Cartilage Degradation and Resulting Interactions with the Complement System.Aging and osteoarthritis.

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Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

http://www.wikidata.org/entity/Q37211903

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article científic

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bilimsel makale

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scientific article published on 28 May 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

@en

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

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type

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Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

@en

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

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prefLabel

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

@en

Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

@nl

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ARD.2009.119966

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Annals of the Rheumatic Diseases

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Superoxide dismutase downregulation in osteoarthritis progression and end-stage disease.

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Christos Gabrielides

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David A Young

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Gillian A Wallis

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Ian M Clark

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Jenny L Scott

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Raymond P Boot-Handford

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Robert W Taylor

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Rose K Davidson

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Tom B L Kirkwood

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Tracey E Swingler

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P2860

Potential involvement of oxidative stress in cartilage senescence and development of osteoarthritis: oxidative stress induces chondrocyte telomere instability and downregulation of chondrocyte function.

Expression profiling of metalloproteinases and their inhibitors in cartilage

Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

Stable overexpression of manganese superoxide dismutase in mitochondria identifies hydrogen peroxide as a major oxidant in the AP-1-mediated induction of matrix-degrading metalloprotease-1

Phenotypes of mice lacking extracellular superoxide dismutase and copper- and zinc-containing superoxide dismutase

Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

BiQ Analyzer: visualization and quality control for DNA methylation data from bisulfite sequencing.

Superoxide, hydrogen peroxide and nitric oxide as signalling molecules: their production and role in disease.

Aging, articular cartilage chondrocyte senescence and osteoarthritis.

The role of reactive oxygen species in homeostasis and degradation of cartilage.

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