Keratin mutation predisposes to mouse liver fibrosis and unmasks differential effects of the carbon tetrachloride and thioacetamide models.
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Toward unraveling the complexity of simple epithelial keratins in human diseaseDifference in expression of hepatic microRNAs miR-29c, miR-34a, miR-155, and miR-200b is associated with strain-specific susceptibility to dietary nonalcoholic steatohepatitis in miceIntermediate filaments take the heat as stress proteins.Hepatic fibrosis.Non-coding keratin variants associate with liver fibrosis progression in patients with hemochromatosis.Keratin 8 variants are infrequent in patients with alcohol-related liver cirrhosis and do not associate with development of hepatocellular carcinomaIdentification of cytokeratin 18 as a biomarker of mouse and human hepatosplenic schistosomiasis.A mouse model of cholestasis-associated cholangiocarcinoma and transcription factors involved in progression.Prevalence of genetic variants of keratins 8 and 18 in patients with drug-induced liver injuryCostaining for keratins 8/18 plus ubiquitin improves detection of hepatocyte injury in nonalcoholic fatty liver disease.Human keratin 8 variants promote mouse acetaminophen hepatotoxicity coupled with c-jun amino-terminal kinase activation and protein adduct formation.Absence of keratin 8 or 18 promotes antimitochondrial autoantibody formation in aging male miceIntermediate filament cytoskeleton of the liver in health and diseaseHigh-Throughput Screening for Drugs that Modulate Intermediate Filament ProteinsRecent advances in understanding the roles of transglutaminase 2 in alcoholic steatohepatitis.Hepatocyte-Specific Deletion of Mouse Lamin A/C Leads to Male-Selective Steatohepatitis.Functional proteomics reveals hepatotoxicity and the molecular mechanisms of different forms of chromium delivered by skin administration.Standard operating procedures in experimental liver research: thioacetamide model in mice and rats.Hepcidin knockout mice spontaneously develop chronic pancreatitis owing to cytoplasmic iron overload in acinar cells.CHOP-mediated hepcidin suppression modulates hepatic iron load.Granulocyte colony stimulating factor treatment in non-alcoholic fatty liver disease: beyond marrow cell mobilization.Loss of keratin 19 favours the development of cholestatic liver disease through decreased ductular reaction.Quantitative chemical proteomics for investigating the biomarkers of dioscin against liver fibrosis caused by CCl4 in rats.
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P2860
Keratin mutation predisposes to mouse liver fibrosis and unmasks differential effects of the carbon tetrachloride and thioacetamide models.
description
article científic
@ca
article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 18 January 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Keratin mutation predisposes t ...... ride and thioacetamide models.
@en
Keratin mutation predisposes t ...... ride and thioacetamide models.
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type
label
Keratin mutation predisposes t ...... ride and thioacetamide models.
@en
Keratin mutation predisposes t ...... ride and thioacetamide models.
@nl
prefLabel
Keratin mutation predisposes t ...... ride and thioacetamide models.
@en
Keratin mutation predisposes t ...... ride and thioacetamide models.
@nl
P2093
P2860
P1433
P1476
Keratin mutation predisposes t ...... oride and thioacetamide models
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P2093
Diana M Toivola
Elizabeth M Brunt
Guo-Zhong Tao
Masaru Harada
Pavel Strnad
P2860
P304
P356
10.1053/J.GASTRO.2008.01.035
P407
P50
P577
2008-01-18T00:00:00Z